KALLIKREIN, KININOGEN AND KININS IN CONTROL OF BLOOD-PRESSURE

被引:75
作者
MILLS, IH
机构
来源
NEPHRON | 1979年 / 23卷 / 2-3期
关键词
D O I
10.1159/000181611
中图分类号
R5 [内科学]; R69 [泌尿科学(泌尿生殖系疾病)];
学科分类号
1002 ; 100201 ;
摘要
Plasma kallikrein releases bradykinin when activated by gram-negative septicemia or irreversible hemorrhagic shock. Pancreatitis releases glandular kallikrein causing hypotension and increased vascular permeability. Bradykinin in the brain produces hypertension. Renal kallikrein is released by high arterial pressure, vasodilators, low doses of noradrenaline, angiotensin II, mineralocorticoids and rapid volume expansion. It has a biphasic relation to sodium excretion. In essential hypertension kallikrein release into blood and urine is low and facilitates hypertension. High renin in Bartter's syndrome is balanced by high PGE and kallikrein without hypertension.
引用
收藏
页码:61 / 71
页数:11
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