OCCUPATIONAL RISK FOR HEPATITIS-C VIRUS-INFECTION AMONG NEW-YORK-CITY DENTISTS

被引:141
作者
KLEIN, RS
FREEMAN, K
TAYLOR, PE
STEVENS, CE
机构
[1] MONTEFIORE MED CTR,DEPT BIOSTAT,BRONX,NY 10467
[2] YESHIVA UNIV ALBERT EINSTEIN COLL MED,DEPT MED,BRONX,NY 10461
[3] YESHIVA UNIV ALBERT EINSTEIN COLL MED,DEPT EPIDEMIOL,BRONX,NY 10461
[4] YESHIVA UNIV ALBERT EINSTEIN COLL MED,DEPT SOCIAL MED,BRONX,NY 10461
[5] NEW YORK BLOOD CTR,WOLF SZMUNESS LAB EPIDEMIOL,NEW YORK,NY 10021
关键词
D O I
10.1016/0140-6736(91)92369-D
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
Health-care workers have an occupational risk of infection with hepatitis C virus (HCV). However, neither the magnitude of this risk nor the practices associated with it have been defined. Since dentists have numerous patients and are exposed to blood, they are likely to have the maximum risk. Therefore, we have assessed occupational risk for HCV infection among dentists in the New York City area. Individuals who admitted present or previous intravenous drug use or (men) who were homosexual or bisexual were excluded. Demographic, occupational, and behavioural data were recorded, and sera were tested for antibodies to HCV (anti-HCV). Anti-HCV was found in 8 (1.75%) of 456 dentists compared with 1 (0.14%) of 723 controls (odds ratio [OR] 12.9, 95% confidence interval [Cl] 1.7 to 573). Anti-HCV was found in 4 (9.3%) of 43 oral surgeons compared with 4 (0.97%) of 413 other dentists (OR 10.5, 95% Cl 1.9 to 58). Seropositive dentists claimed to have treated more intravenous drug users in the week (p = 0.04) or month (p = 0.03) before the study than did seronegative dentists. Our findings show that dentists are at increased risk for hepatitis C infection. All health-care workers should regard patients as potentially infected with a communicable bloodborne agent.
引用
收藏
页码:1539 / 1542
页数:4
相关论文
共 28 条
[1]   HEPATITIS-C VIRUS-INFECTION IN POSTTRANSFUSION HEPATITIS - AN ANALYSIS WITH 1ST-GENERATION AND 2ND-GENERATION ASSAYS [J].
AACH, RD ;
STEVENS, CE ;
HOLLINGER, FB ;
MOSLEY, JW ;
PETERSON, DA ;
TAYLOR, PE ;
JOHNSON, RG ;
BARBOSA, LH ;
NEMO, GJ .
NEW ENGLAND JOURNAL OF MEDICINE, 1991, 325 (19) :1325-1329
[2]   EXPERIMENTAL TRANSMISSION OF NON-A, NON-B HEPATITIS BY SALIVA [J].
ABE, K ;
KURATA, T ;
SHIKATA, T ;
SUGITANI, M ;
ODA, T .
JOURNAL OF INFECTIOUS DISEASES, 1987, 155 (05) :1078-1079
[3]  
AHTONE J, 1980, LANCET, V1, P1142
[4]   DETECTION OF ANTIBODY TO HEPATITIS-C VIRUS IN PROSPECTIVELY FOLLOWED TRANSFUSION RECIPIENTS WITH ACUTE AND CHRONIC NON-A-HEPATITIS, NON-B-HEPATITIS [J].
ALTER, HJ ;
PURCELL, RH ;
SHIH, JW ;
MELPOLDER, JC ;
HOUGHTON, M ;
CHOO, QL ;
KUO, G .
NEW ENGLAND JOURNAL OF MEDICINE, 1989, 321 (22) :1494-1500
[5]  
ALTER HJ, 1978, LANCET, V1, P459
[6]   THE EFFECT OF UNDERREPORTING ON THE APPARENT INCIDENCE AND EPIDEMIOLOGY OF ACUTE VIRAL-HEPATITIS [J].
ALTER, MJ ;
MARES, A ;
HADLER, SC ;
MAYNARD, JE .
AMERICAN JOURNAL OF EPIDEMIOLOGY, 1987, 125 (01) :133-139
[7]   SPORADIC NON-A, NON-B HEPATITIS - FREQUENCY AND EPIDEMIOLOGY IN AN URBAN UNITED-STATES POPULATION [J].
ALTER, MJ ;
GERETY, RJ ;
SMALLWOOD, LA ;
SAMPLINER, RE ;
TABOR, E ;
DEINHARDT, F ;
FROSNER, G ;
MATANOSKI, GM .
JOURNAL OF INFECTIOUS DISEASES, 1982, 145 (06) :886-893
[8]   RISK-FACTORS FOR ACUTE NON-A, NON-B HEPATITIS IN THE UNITED-STATES AND ASSOCIATION WITH HEPATITIS-C VIRUS-INFECTION [J].
ALTER, MJ ;
HADLER, SC ;
JUDSON, FN ;
MARES, A ;
ALEXANDER, WJ ;
HU, PY ;
MILLER, JK ;
MOYER, LA ;
FIELDS, HA ;
BRADLEY, DW ;
MARGOLIS, HS .
JAMA-JOURNAL OF THE AMERICAN MEDICAL ASSOCIATION, 1990, 264 (17) :2231-2235
[9]   IMPORTANCE OF HETEROSEXUAL ACTIVITY IN THE TRANSMISSION OF HEPATITIS-B AND NON-A, NON-B HEPATITIS [J].
ALTER, MJ ;
COLEMAN, PJ ;
ALEXANDER, WJ ;
KRAMER, E ;
MILLER, JK ;
MANDEL, E ;
HADLER, SC ;
MARGOLIS, HS .
JAMA-JOURNAL OF THE AMERICAN MEDICAL ASSOCIATION, 1989, 262 (09) :1201-1205
[10]  
BRUIX J, 1989, LANCET, V2, P1004