ESCHERICHIA-COLI STRAINS WITH MULTIPLE DNA-REPAIR DEFECTS ARE HYPERINDUCED FOR THE SOS RESPONSE

被引：8

作者：

FOSTER, PL ^{[1
]}

机构：

[1] BOSTON UNIV, SCH MED, DEPT PATHOL, BOSTON, MA 02118 USA

来源：

JOURNAL OF BACTERIOLOGY | 1990年 / 172卷 / 08期

关键词：

D O I：

10.1128/jb.172.8.4719-4720.1990

中图分类号：

Q93 [微生物学];

学科分类号：

071005 ; 100705 ;

摘要：

Escherichia coli strains defective for the repair of apurinic/apyrimidinic sites and for the UvrABC excision repair pathway could be constructed if they also carried a mutation in ung, which encodes uracil glycosylase or sulA, which encodes an SOS-inducible inhibitor of septation. The results strains were sensitive to alkylation damage and hyperinduced for the SOS response, but had unpredictable spontaneous mutation rates.

引用

页码：4719 / 4720

页数：2

共 12 条

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