CA-2+-DEPENDENT K+ CHANNELS IN NEURO-BLASTOMA HYBRID-CELLS ACTIVATED BY INTRACELLULAR INOSITOL TRISPHOSPHATE AND EXTRACELLULAR BRADYKININ

被引：31

作者：

HIGASHIDA, H

BROWN, DA

机构：

[1] NHLBI,BIOCHEM GENET LAB,BETHESDA,MD 20892

[2] NIH,FOGARTY INT CTR,BETHESDA,MD 20892

来源：

FEBS LETTERS | 1988年 / 238卷 / 02期

关键词：

D O I：

10.1016/0014-5793(88)80519-2

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

引用

页码：395 / 400

页数：6

共 24 条

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[3]

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HIGASHIDA, H .

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[6]

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[7]

DOWNES CP, 1985, MOL MECHANISMS TRANS, P3

[8] PROPERTIES OF A CA-2+ ACTIVATED K+-CONDUCTANCE IN HELIX NEURONS INVESTIGATED BY INTRACELLULAR CA-2+ IONOPHORESIS [J].

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[9] BRADYKININ INHIBITS POTASSIUM (M) CURRENTS IN N1E-115 NEUROBLASTOMA-CELLS - RESPONSES RESEMBLE THOSE IN NG108-15 NEUROBLASTOMA X GLIOMA HYBRID-CELLS [J].

HIGASHIDA, H ;

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FEBS LETTERS, 1987, 220 (02) :302-306

[10] BRADYKININ-ACTIVATED TRANSMEMBRANE SIGNALS ARE COUPLED VIA NO OR NI TO PRODUCTION OF INOSITOL 1,4,5-TRISPHOSPHATE, A 2ND MESSENGER IN NG108-15 NEUROBLASTOMA GLIOMA HYBRID-CELLS [J].

HIGASHIDA, H ;

STREATY, RA ;

KLEE, W ;

NIRENBERG, M .

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1986, 83 (04) :942-946

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