DIFFERENT STRUCTURAL ALTERATIONS UPREGULATE INVITRO TYROSINE KINASE-ACTIVITY AND TRANSFORMING POTENCY OF THE ERBB-2 GENE

被引：177

作者：

SEGATTO, O ^{[1
]}

KING, CR ^{[1
]}

PIERCE, JH ^{[1
]}

DIFIORE, PP ^{[1
]}

AARONSON, SA ^{[1
]}

机构：

[1] NCI,CELLULAR & MOLEC BIOL LAB,BLDG 37,ROOM 1E24,BETHESDA,MD 20892

来源：

MOLECULAR AND CELLULAR BIOLOGY | 1988年 / 8卷 / 12期

关键词：

D O I：

10.1128/MCB.8.12.5570

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

引用

页码：5570 / 5574

页数：5

共 22 条

[1] THE PRODUCT OF THE HUMAN C-ERBB-2 GENE - A 185-KILODALTON GLYCOPROTEIN WITH TYROSINE KINASE-ACTIVITY
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[2] THE NEU ONCOGENE ENCODES AN EPIDERMAL GROWTH-FACTOR RECEPTOR-RELATED PROTEIN
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[J]. NATURE, 1986, 319 (6050) : 226 - 230
[3] MULTIPLE INDEPENDENT ACTIVATIONS OF THE NEU ONCOGENE BY A POINT MUTATION ALTERING THE TRANSMEMBRANE DOMAIN OF P185
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[J]. CELL, 1986, 45 (05) : 649 - 657
[4] TYROSINE KINASE RECEPTOR WITH EXTENSIVE HOMOLOGY TO EGF RECEPTOR SHARES CHROMOSOMAL LOCATION WITH NEU ONCOGENE
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[J]. SCIENCE, 1985, 230 (4730) : 1132 - 1139
[5] ERBB-2 IS A POTENT ONCOGENE WHEN OVEREXPRESSED IN NIH/3T3 CELLS
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SEGATTO, O
KING, CR
AARONSON, SA
[J]. SCIENCE, 1987, 237 (4811) : 178 - 182
[6] INCREASED EXPRESSION OF THE PUTATIVE GROWTH-FACTOR RECEPTOR P185HER2 CAUSES TRANSFORMATION AND TUMORIGENESIS OF NIH-3T3 CELLS
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ULLRICH, A
[J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1987, 84 (20) : 7159 - 7163
[7] AMPLIFICATION OF A NOVEL V-ERBB-RELATED GENE IN A HUMAN MAMMARY-CARCINOMA
KING, CR
KRAUS, MH
AARONSON, SA
[J]. SCIENCE, 1985, 229 (4717) : 974 - 976
[8] OVEREXPRESSION OF THE EGF RECEPTOR-RELATED PROTOONCOGENE ERBB-2 IN HUMAN MAMMARY-TUMOR CELL-LINES BY DIFFERENT MOLECULAR MECHANISMS
KRAUS, MH
POPESCU, NC
AMSBAUGH, SC
KING, CR
[J]. EMBO JOURNAL, 1987, 6 (03) : 605 - 610
[9] RAPID AND EFFICIENT SITE-SPECIFIC MUTAGENESIS WITHOUT PHENOTYPIC SELECTION
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[J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1985, 82 (02) : 488 - 492
[10] LELAND E, 1987, MOL ENDOCRINOL, V1, P15

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