EFFECT OF INFLAMMATORY AND NON-INFLAMMATORY STRESS ON PLASMA KETONE-BODIES AND FREE FATTY-ACIDS AND ON GLUCAGON AND INSULIN IN PERIPHERAL AND PORTAL BLOOD

被引：18

作者：

KAMINSKI, MV ^{[1
]}

NEUFELD, HA ^{[1
]}

PACE, JG ^{[1
]}

机构：

[1] ST MARY NAZARETH HOSP CTR,CHICAGO,IL 60622

来源：

INFLAMMATION | 1979年 / 3卷 / 03期

关键词：

D O I：

10.1007/BF00914186

中图分类号：

Q2 [细胞生物学];

学科分类号：

071009 ; 090102 ;

摘要：

Inflammatory stress as characterized by infection with Streptococcus pneumoniae, administration of endotoxin, or the induction of a turpentine abscess is characterized by the inhibition of the ketosis associated with fasting and a decline in the level of free fatty acids in the plasma. Moreover, rats subjected to these inflammatory stresses demonstrate a significant rise in peripheral and portal insulin and glucagon. Rats subjected to noninflammatory stresses, screen-restraint, or noninvasive femoral fracture did not demonstrate the inhibition of ketosis but did show a decrease in plasma free fatty acids. The noninflammatory stresses did not show an abnormal elevation of plasma or portal insulin or glucagon. © 1979 Plenum Publishing Corporation.

引用

页码：289 / 294

页数：6

共 18 条

[1] FATTY-ACID AND KETONE-BODY METABOLISM IN RAT - RESPONSE TO DIET AND EXERCISE [J].