MRI DEMONSTRATION OF CORTICAL LAMINAR NECROSIS AND DELAYED WHITE MATTER INJURY IN ANOXIC ENCEPHALOPATHY

被引：87

作者：

SAWADA, H ^{[1
]}

UDAKA, F ^{[1
]}

SERIU, N ^{[1
]}

SHINDOU, K ^{[1
]}

KAMEYAMA, M ^{[1
]}

TSUJIMURA, M ^{[1
]}

机构：

[1] SUMITOMO HOSP,DEPT PATHOL,OSAKA 530,JAPAN

来源：

NEURORADIOLOGY | 1990年 / 32卷 / 04期

关键词：

Anoxic encephalopathy; Cortical laminar necrosis; Delayed-onset progressive white matter lesion; MRI;

D O I：

10.1007/BF00593053

中图分类号：

R74 [神经病学与精神病学];

学科分类号：

摘要：

We performed serial radiological examinations on a patient with anoxic encephalopathy. In the early term after the anoxic insult, T1-weighted MRI revealed high signal intensity areas distributed laminarly in the cerebral cortex and diffusely in the putamen, which were thought to refect the cortical necrosis and necrosis in the putamen. Single photon emission computed tomography using I-123 isopropylamphetamine showed persistent hypoperfusion in the arterial watershed zones. T2-weighted MRI performed several months after the anoxic episode revealed diffuse high-intensity lesions in the arterial water-shed zones. These delayed-onset white matter lesions continued to extend over several months. © 1990 Springer-Verlag.

引用

页码：319 / 321

页数：3

共 13 条

[1]

[Anonymous], [No title captured]

[2]

BRIERLEY JB, 1984, GREENFIELDS NEUROPAT, P125

[3] HYPOXIC-ISCHEMIC LEUKOENCEPHALOPATHY IN MAN [J].