DECREASED SYMPATHOCHROMAFFIN ACTIVITY IN IDDM

[1] DECREASED ALPHA-2-ADRENERGIC RECEPTORS ON PLATELET MEMBRANES FROM DIABETIC-PATIENTS WITH AUTONOMIC NEUROPATHY AND ORTHOSTATIC HYPOTENSION [J]. JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM, 1986, 63 (04) : 906 - 912

[2] METABOLIC HOMEOSTASIS IN JUVENILE DIABETES MELLLITUS .2. INCREASED KETONE RESPONSIVENESS TO EPINEPHRINE [J]. DIABETES, 1969, 18 (06) : 421 - &

[3] ENHANCED GLYCEMIC RESPONSIVENESS TO EPINEPHRINE IN INSULIN-DEPENDENT DIABETES-MELLITUS IS THE RESULT OF THE INABILITY TO SECRETE INSULIN - AUGMENTED INSULIN-SECRETION NORMALLY LIMITS THE GLYCEMIC, BUT NOT THE LIPOLYTIC OR KETOGENIC, RESPONSE TO EPINEPHRINE IN HUMANS [J]. JOURNAL OF CLINICAL INVESTIGATION, 1985, 75 (06) : 1842 - 1851

[4] DECREASED BETA-ADRENERGIC SENSITIVITY IN INSULIN-DEPENDENT DIABETIC SUBJECTS [J]. JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM, 1986, 63 (01) : 262 - 265

[5] BERRETTAPICCOLI C, 1981, AM J MED, V71, P829

[6] BEYLOT M, 1985, DIABETES METAB, V11, P111

[7] A RELIABLE AND REPRODUCIBLE TEST FOR ADEQUATE GLUCOSE COUNTERREGULATION IN TYPE-I DIABETES-MELLITUS [J]. DIABETES, 1984, 33 (08) : 732 - 737

[8] ALPHA-ADRENERGIC BLOCKADE IMPROVES GLUCOSE-POTENTIATED INSULIN-SECRETION IN NON-INSULIN-DEPENDENT DIABETES-MELLITUS [J]. DIABETES, 1987, 36 (08) : 932 - 937

[9] PLASMA CATECHOLAMINES IN LONG-TERM DIABETICS WITH AND WITHOUT NEUROPATHY AND IN HYPOPHYSECTOMIZED SUBJECTS [J]. JOURNAL OF CLINICAL INVESTIGATION, 1972, 51 (04) : 779 - +

[10] CHRISTENSEN NJ, 1979, DIABETOLOGIA, V16, P211, DOI 10.1007/BF01221946

[1] DECREASED ALPHA-2-ADRENERGIC RECEPTORS ON PLATELET MEMBRANES FROM DIABETIC-PATIENTS WITH AUTONOMIC NEUROPATHY AND ORTHOSTATIC HYPOTENSION [J]. JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM, 1986, 63 (04) : 906 - 912

[2] METABOLIC HOMEOSTASIS IN JUVENILE DIABETES MELLLITUS .2. INCREASED KETONE RESPONSIVENESS TO EPINEPHRINE [J]. DIABETES, 1969, 18 (06) : 421 - &

[3] ENHANCED GLYCEMIC RESPONSIVENESS TO EPINEPHRINE IN INSULIN-DEPENDENT DIABETES-MELLITUS IS THE RESULT OF THE INABILITY TO SECRETE INSULIN - AUGMENTED INSULIN-SECRETION NORMALLY LIMITS THE GLYCEMIC, BUT NOT THE LIPOLYTIC OR KETOGENIC, RESPONSE TO EPINEPHRINE IN HUMANS [J]. JOURNAL OF CLINICAL INVESTIGATION, 1985, 75 (06) : 1842 - 1851

[4] DECREASED BETA-ADRENERGIC SENSITIVITY IN INSULIN-DEPENDENT DIABETIC SUBJECTS [J]. JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM, 1986, 63 (01) : 262 - 265

[5] BERRETTAPICCOLI C, 1981, AM J MED, V71, P829

[6] BEYLOT M, 1985, DIABETES METAB, V11, P111

[7] A RELIABLE AND REPRODUCIBLE TEST FOR ADEQUATE GLUCOSE COUNTERREGULATION IN TYPE-I DIABETES-MELLITUS [J]. DIABETES, 1984, 33 (08) : 732 - 737

[8] ALPHA-ADRENERGIC BLOCKADE IMPROVES GLUCOSE-POTENTIATED INSULIN-SECRETION IN NON-INSULIN-DEPENDENT DIABETES-MELLITUS [J]. DIABETES, 1987, 36 (08) : 932 - 937

[9] PLASMA CATECHOLAMINES IN LONG-TERM DIABETICS WITH AND WITHOUT NEUROPATHY AND IN HYPOPHYSECTOMIZED SUBJECTS [J]. JOURNAL OF CLINICAL INVESTIGATION, 1972, 51 (04) : 779 - +

[10] CHRISTENSEN NJ, 1979, DIABETOLOGIA, V16, P211, DOI 10.1007/BF01221946