MOLECULAR ADVANCES IN CARDIOVASCULAR BIOLOGY

[1] DELETION POLYMORPHISM IN THE GENE FOR ANGIOTENSIN-CONVERTING ENZYME IS A POTENT RISK FACTOR FOR MYOCARDIAL-INFARCTION [J]. NATURE, 1992, 359 (6396) : 641 - 644

[2] CHIEN KR, 1993, ANNU REV PHYSIOL, V55, P77, DOI 10.1146/annurev.physiol.55.1.77

[3] REGULATION OF CARDIAC GENE-EXPRESSION DURING MYOCARDIAL GROWTH AND HYPERTROPHY - MOLECULAR STUDIES OF AN ADAPTIVE PHYSIOLOGICAL-RESPONSE [J]. FASEB JOURNAL, 1991, 5 (15) : 3037 - 3046

[4] REGIONALLY RESTRICTED DEVELOPMENTAL DEFECTS RESULTING FROM TARGETED DISRUPTION OF THE MOUSE HOMEOBOX GENE HOX-1.5 [J]. NATURE, 1991, 350 (6318) : 473 - 479

[5] CSERJESI P, 1992, DEVELOPMENT, V115, P1087

[6] SPLOTCH (SP2H), A MUTATION AFFECTING DEVELOPMENT OF THE MOUSE NEURAL-TUBE, SHOWS A DELETION WITHIN THE PAIRED HOMEODOMAIN OF PAX-3 [J]. CELL, 1991, 67 (04) : 767 - 774

[7] FARRANCE IKG, 1992, J BIOL CHEM, V267, P17234

[8] INHIBITION OF NEOINTIMAL SMOOTH-MUSCLE ACCUMULATION AFTER ANGIOPLASTY BY AN ANTIBODY TO PDGF [J]. SCIENCE, 1991, 253 (5024) : 1129 - 1132

[9] ATRIAL NATRIURETIC FACTOR-SV40 T-ANTIGEN TRANSGENES PRODUCE TUMORS AND CARDIAC-ARRHYTHMIAS IN MICE [J]. SCIENCE, 1988, 239 (4843) : 1029 - 1033

[10] A MOLECULAR-BASIS FOR FAMILIAL HYPERTROPHIC CARDIOMYOPATHY - A BETA-CARDIAC MYOSIN HEAVY-CHAIN GENE MISSENSE MUTATION [J]. CELL, 1990, 62 (05) : 999 - 1006

[1] DELETION POLYMORPHISM IN THE GENE FOR ANGIOTENSIN-CONVERTING ENZYME IS A POTENT RISK FACTOR FOR MYOCARDIAL-INFARCTION [J]. NATURE, 1992, 359 (6396) : 641 - 644

[2] CHIEN KR, 1993, ANNU REV PHYSIOL, V55, P77, DOI 10.1146/annurev.physiol.55.1.77

[3] REGULATION OF CARDIAC GENE-EXPRESSION DURING MYOCARDIAL GROWTH AND HYPERTROPHY - MOLECULAR STUDIES OF AN ADAPTIVE PHYSIOLOGICAL-RESPONSE [J]. FASEB JOURNAL, 1991, 5 (15) : 3037 - 3046

[4] REGIONALLY RESTRICTED DEVELOPMENTAL DEFECTS RESULTING FROM TARGETED DISRUPTION OF THE MOUSE HOMEOBOX GENE HOX-1.5 [J]. NATURE, 1991, 350 (6318) : 473 - 479

[5] CSERJESI P, 1992, DEVELOPMENT, V115, P1087

[6] SPLOTCH (SP2H), A MUTATION AFFECTING DEVELOPMENT OF THE MOUSE NEURAL-TUBE, SHOWS A DELETION WITHIN THE PAIRED HOMEODOMAIN OF PAX-3 [J]. CELL, 1991, 67 (04) : 767 - 774

[7] FARRANCE IKG, 1992, J BIOL CHEM, V267, P17234

[8] INHIBITION OF NEOINTIMAL SMOOTH-MUSCLE ACCUMULATION AFTER ANGIOPLASTY BY AN ANTIBODY TO PDGF [J]. SCIENCE, 1991, 253 (5024) : 1129 - 1132

[9] ATRIAL NATRIURETIC FACTOR-SV40 T-ANTIGEN TRANSGENES PRODUCE TUMORS AND CARDIAC-ARRHYTHMIAS IN MICE [J]. SCIENCE, 1988, 239 (4843) : 1029 - 1033

[10] A MOLECULAR-BASIS FOR FAMILIAL HYPERTROPHIC CARDIOMYOPATHY - A BETA-CARDIAC MYOSIN HEAVY-CHAIN GENE MISSENSE MUTATION [J]. CELL, 1990, 62 (05) : 999 - 1006