DEXAMETHASONE-SUPPRESSIBLE HYPERALDOSTERONISM - ADRENAL TRANSITION CELL HYPERPLASIA

被引：44

作者：

CONNELL, JMC ^{[1
]}

KENYON, CJ ^{[1
]}

CORRIE, JET ^{[1
]}

FRASER, R ^{[1
]}

WATT, R ^{[1
]}

LEVER, AF ^{[1
]}

机构：

[1] WESTERN GEN HOSP,MRC,POPULAT & CYTOGENET UNIT,EDINBURGH EH4 2XU,MIDLOTHIAN,SCOTLAND

来源：

HYPERTENSION | 1986年 / 8卷 / 08期

关键词：

D O I：

10.1161/01.HYP.8.8.669

中图分类号：

R6 [外科学];

学科分类号：

1002 ; 100210 ;

摘要：

引用

页码：669 / 676

页数：8

共 33 条

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[3] CONCURRENT ESTIMATION OF TOTAL-BODY AND EXCHANGEABLE BODY SODIUM IN HYPERTENSION
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[4] DOPAMINERGIC INHIBITION OF METOCLOPRAMIDE-INDUCED ALDOSTERONE SECRETION IN MAN - DISSOCIATION OF RESPONSES TO DOPAMINE AND BROMOCRIPTINE
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[5] CONNELL JMC, 1986, J ENDOCRINOL
[6] CORRIE JET, 1985, CLIN CHEM, V31, P849
[7] METYRAPONE AND ANTIOXIDANTS ARE REQUIRED TO MAINTAIN ALDOSTERONE SYNTHESIS BY CULTURED BOVINE ADRENOCORTICAL ZONA GLOMERULOSA CELLS
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[8] INDUCTION OF CULTURED BOVINE ADRENOCORTICAL ZONA GLOMERULOSA CELL 17-HYDROXYLASE ACTIVITY BY ACTH
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[9] ESTIMATION OF ANGIOTENSIN II CONCENTRATION IN HUMAN PLASMA BY RADIOIMMUNOASSAY - SOME APPLICATIONS TO PHYSIOLOGICAL AND CLINICAL STATES
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[J]. EUROPEAN JOURNAL OF CLINICAL INVESTIGATION, 1971, 2 (01) : 32 - +
[10] A NEW FAMILY WITH DEXAMETHASONE-SUPPRESSIBLE HYPER-ALDOSTERONISM - ALDOSTERONE UNRESPONSIVENESS TO ANGIOTENSIN-II
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