LOSS OF OMPC PORIN IN A STRAIN OF SALMONELLA-TYPHIMURIUM CAUSES INCREASED RESISTANCE TO CEPHALOSPORINS DURING THERAPY

被引：62

作者：

MEDEIROS, AA

OBRIEN, TF

ROSENBERG, EY

NIKAIDO, H

机构：

[1] BROWN UNIV,PROVIDENCE,RI 02912

[2] BRIGHAM & WOMENS HOSP,DEPT MED,BOSTON,MA 02115

[3] HARVARD UNIV,SCH MED,DEPT MICROBIOL & IMMUNOL,BOSTON,MA 02115

来源：

JOURNAL OF INFECTIOUS DISEASES | 1987年 / 156卷 / 05期

关键词：

D O I：

10.1093/infdis/156.5.751

中图分类号：

R392 [医学免疫学]; Q939.91 [免疫学];

学科分类号：

100102 ;

摘要：

引用

页码：751 / 757

页数：7

共 27 条

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[3] PLEIOTROPIC TRANSPORT MUTANTS OF ESCHERICHIA-COLI LACK PORIN, A MAJOR OUTER MEMBRANE-PROTEIN
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[7] MUTANTS OF ESCHERICHIA-COLI THAT ARE RESISTANT TO CERTAIN BETA-LACTAM COMPOUNDS LACK THE OMPF PORIN
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[J]. ANTIMICROBIAL AGENTS AND CHEMOTHERAPY, 1981, 20 (04) : 549 - 552
[8] ALTERATIONS IN OUTER MEMBRANE PROTEINS OF ESCHERICHIA-COLI B-R ASSOCIATED WITH PRESENCE OF R-PLASMID RRM98
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[J]. FEBS LETTERS, 1978, 85 (01): : 127 - 132
[9] ROLE OF PORIN PROTEINS OMPF AND OMPC IN THE PERMEATION OF BETA-LACTAMS
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[J]. ANTIMICROBIAL AGENTS AND CHEMOTHERAPY, 1982, 22 (06) : 942 - 948
[10] ANALYTICAL ISOELECTRIC FOCUSING OF R-FACTOR-DETERMINED BETA-LACTAMASES - CORRELATION WITH PLASMID COMPATIBILITY
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