GENETIC-CONTROL OF LIVER CAMP LEVELS IN MICE

被引：16

作者：

LAFUSE, W

MERUELO, D

EDIDIN, M

机构：

[1] JOHNS HOPKINS UNIV,DEPT BIOL,BALTIMORE,MD 21218

[2] MAYO CLIN & MAYO FDN,DEPT IMMUNOL,ROCHESTER,MN 55901

[3] NYU MED CTR,MED CTR,DEPT PATHOL,IRVINGTON HOUSE INST,NEW YORK,NY 10016

来源：

IMMUNOGENETICS | 1979年 / 9卷 / 01期

关键词：

D O I：

10.1007/BF01570393

中图分类号：

Q3 [遗传学];

学科分类号：

071007 ; 090102 ;

摘要：

Steady-state level of liver 3′,5′-cyclic monophosphate, cAMP, has been shown to be under genetic control linked to the mouse H-2 complex. Liver cAMP levels are associated with H-2 haplotype in fully segregating crosses of strains C3H and C57BL/10. In crosses involving strain A, other loci have an effect that swamps that of H-2. Results with H-2 recombinants indicate that liver cAMP levels are affected by more than one H-2-linked locus. © 1979 Springer-Verlag New York Inc.

引用

页码：57 / 65

页数：9

共 22 条

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ALTMAN RA, 1979, INBRED GENETICALLY 1, P16

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