FUNCTIONAL MODULATION OF THE NICOTINIC ACETYLCHOLINE-RECEPTOR BY TYROSINE PHOSPHORYLATION

被引：248

作者：

HOPFIELD, JF

TANK, DW

GREENGARD, P

HUGANIR, RL

机构：

[1] ROCKEFELLER UNIV,MOLEC & CELLULAR NEUROSCI LAB,NEW YORK,NY 10021

[2] AT&T BELL LABS,MOLEC BIOPHYS RES DEPT,MURRAY HILL,NJ 07974

来源：

NATURE | 1988年 / 336卷 / 6200期

关键词：

D O I：

10.1038/336677a0

中图分类号：

O [数理科学和化学]; P [天文学、地球科学]; Q [生物科学]; N [自然科学总论];

学科分类号：

07 ; 0710 ; 09 ;

摘要：

引用

页码：677 / 680

页数：4

共 30 条

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[J]. MOLECULAR AND CELLULAR BIOLOGY, 1983, 3 (06) : 1157 - 1162
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[6] AGENTS THAT ACTIVATE PROTEIN KINASE-C REDUCE ACETYLCHOLINE SENSITIVITY IN CULTURED MYOTUBES
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[7] MULTIPLE CONDUCTANCE STATES OF SINGLE ACETYLCHOLINE-RECEPTOR CHANNELS IN EMBRYONIC MUSCLE-CELLS
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SAKMANN, B
[J]. NATURE, 1981, 294 (5840) : 462 - 464
[8] PROTEIN TYROSINE KINASE-ACTIVITY AND ITS ENDOGENOUS SUBSTRATES IN RAT-BRAIN - A SUBCELLULAR AND REGIONAL SURVEY
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GREENGARD, P
HUGANIR, RL
[J]. JOURNAL OF NEUROCHEMISTRY, 1988, 50 (05) : 1447 - 1455
[9] PHOSPHORYLATION OF THE NICOTINIC ACETYLCHOLINE-RECEPTOR REGULATES ITS RATE OF DESENSITIZATION
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HESS, GP
[J]. NATURE, 1986, 321 (6072) : 774 - 776
[10] HUGANIR RL, 1982, J BIOL CHEM, V257, P9372

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