INTERACTION WITH NORMAL-CELLS SUPPRESSES THE TRANSFORMED PHENOTYPE OF V-MYC-TRANSFORMED QUAIL MUSCLE-CELLS

被引：61

作者：

LAROCCA, SA ^{[1
]}

GROSSI, M ^{[1
]}

FALCONE, G ^{[1
]}

ALEMA, S ^{[1
]}

TATO, F ^{[1
]}

机构：

[1] CNR,IST BIOL CELLULARE,I-00196 ROME,ITALY

来源：

CELL | 1989年 / 58卷 / 01期

关键词：

D O I：

10.1016/0092-8674(89)90409-1

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

引用

页码：123 / 131

页数：9

共 43 条

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[3] INTERCELLULAR COMMUNICATION AND THE CONTROL OF GROWTH .10. ALTERATION OF JUNCTIONAL PERMEABILITY BY THE SRC GENE - A STUDY WITH TEMPERATURE-SENSITIVE MUTANT ROUS-SARCOMA VIRUS
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[5] BIGNAMI M, 1988, ONCOGENE, V2, P509
[6] SPECIFIC VIRAL ONCOGENES CAUSE DIFFERENTIAL-EFFECTS ON CELL-TO-CELL COMMUNICATION, RELEVANT TO THE SUPPRESSION OF THE TRANSFORMED PHENOTYPE BY NORMAL-CELLS
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[7] DEFECTIVENESS OF AVIAN MYELOCYTOMATOSIS VIRUS MC29 - ISOLATION OF LONG-TERM NONPRODUCER CULTURES AND ANALYSIS OF VIRUS-SPECIFIC POLYPEPTIDE-SYNTHESIS
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[8] THE V-MYC ONCOGENE IS SUFFICIENT TO INDUCE GROWTH TRANSFORMATION OF CHICK NEURORETINA CELLS
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[J]. NATURE, 1987, 326 (6109) : 188 - 190
[9] POTENTIAL ROLE OF THE SRC GENE-PRODUCT IN INHIBITION OF GAP-JUNCTIONAL COMMUNICATION IN NIH/3T3 CELLS
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[J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1985, 82 (16) : 5360 - 5364
[10] NUCLEAR-LOCALIZATION AND DNA-BINDING OF THE TRANSFORMING GENE-PRODUCT OF AVIAN MYELOCYTOMATOSIS VIRUS
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[J]. NATURE, 1982, 296 (5854) : 262 - 266

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