G-PROTEIN-COUPLED RECEPTORS - TURNED ON TO ILL EFFECT

[1] G-PROTEIN-COUPLED RECEPTOR GENES AS PROTOONCOGENES - CONSTITUTIVELY ACTIVATING MUTATION OF THE ALPHA-1B-ADRENERGIC RECEPTOR ENHANCES MITOGENESIS AND TUMORIGENICITY [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1991, 88 (24) : 11354 - 11358

[2] SPECIFIC ACTIVATION OF GS BY SYNTHETIC PEPTIDES CORRESPONDING TO AN INTRACELLULAR LOOP OF THE BETA-ADRENERGIC-RECEPTOR [J]. FEBS LETTERS, 1991, 279 (02) : 277 - 280

[3] REGIONS OF THE ALPHA-1-ADRENERGIC RECEPTOR INVOLVED IN COUPLING TO PHOSPHATIDYLINOSITOL HYDROLYSIS AND ENHANCED SENSITIVITY OF BIOLOGICAL FUNCTION [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1990, 87 (08) : 2896 - 2900

[4] ECTOPIC EXPRESSION OF THE SEROTONIN 1C RECEPTOR AND THE TRIGGERING OF MALIGNANT TRANSFORMATION [J]. SCIENCE, 1989, 244 (4908) : 1057 - 1062

[5] KJELSBERG MA, 1992, J BIOL CHEM, V267, P1430

[6] CONSTITUTIVE ACTIVITY OF RECEPTORS COUPLED TO GUANINE-NUCLEOTIDE REGULATORY PROTEINS [J]. TRENDS IN PHARMACOLOGICAL SCIENCES, 1993, 14 (08) : 303 - 307

[7] OKAMOTO T, 1991, CELL, V67, P1

[8] SOMATIC MUTATIONS IN THE THYROTROPIN RECEPTOR GENE CAUSE HYPERFUNCTIONING THYROID ADENOMAS [J]. NATURE, 1993, 365 (6447) : 649 - 651

[9] REN Q, 1993, J BIOL CHEM, V268, P16483

[10] PIGMENTATION PHENOTYPES OF VARIANT EXTENSION LOCUS ALLELES RESULT FROM POINT MUTATIONS THAT ALTER MSH RECEPTOR FUNCTION [J]. CELL, 1993, 72 (06) : 827 - 834

[1] G-PROTEIN-COUPLED RECEPTOR GENES AS PROTOONCOGENES - CONSTITUTIVELY ACTIVATING MUTATION OF THE ALPHA-1B-ADRENERGIC RECEPTOR ENHANCES MITOGENESIS AND TUMORIGENICITY [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1991, 88 (24) : 11354 - 11358

[2] SPECIFIC ACTIVATION OF GS BY SYNTHETIC PEPTIDES CORRESPONDING TO AN INTRACELLULAR LOOP OF THE BETA-ADRENERGIC-RECEPTOR [J]. FEBS LETTERS, 1991, 279 (02) : 277 - 280

[3] REGIONS OF THE ALPHA-1-ADRENERGIC RECEPTOR INVOLVED IN COUPLING TO PHOSPHATIDYLINOSITOL HYDROLYSIS AND ENHANCED SENSITIVITY OF BIOLOGICAL FUNCTION [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1990, 87 (08) : 2896 - 2900

[4] ECTOPIC EXPRESSION OF THE SEROTONIN 1C RECEPTOR AND THE TRIGGERING OF MALIGNANT TRANSFORMATION [J]. SCIENCE, 1989, 244 (4908) : 1057 - 1062

[5] KJELSBERG MA, 1992, J BIOL CHEM, V267, P1430

[6] CONSTITUTIVE ACTIVITY OF RECEPTORS COUPLED TO GUANINE-NUCLEOTIDE REGULATORY PROTEINS [J]. TRENDS IN PHARMACOLOGICAL SCIENCES, 1993, 14 (08) : 303 - 307

[7] OKAMOTO T, 1991, CELL, V67, P1

[8] SOMATIC MUTATIONS IN THE THYROTROPIN RECEPTOR GENE CAUSE HYPERFUNCTIONING THYROID ADENOMAS [J]. NATURE, 1993, 365 (6447) : 649 - 651

[9] REN Q, 1993, J BIOL CHEM, V268, P16483

[10] PIGMENTATION PHENOTYPES OF VARIANT EXTENSION LOCUS ALLELES RESULT FROM POINT MUTATIONS THAT ALTER MSH RECEPTOR FUNCTION [J]. CELL, 1993, 72 (06) : 827 - 834