CALCIUM-CHANNEL ALTERATIONS IN GENETIC-HYPERTENSION

被引：25

作者：

HERMSMEYER, K

RUSCH, NJ

机构：

[1] OREGON HLTH SCI UNIV,DEPT MED,PORTLAND,OR 97201

[2] OREGON HLTH SCI UNIV,DEPT CELL BIOL,PORTLAND,OR 97201

[3] MED COLL WISCONSIN,DEPT PHYSIOL,MILWAUKEE,WI 53226

来源：

HYPERTENSION | 1989年 / 14卷 / 04期

关键词：

D O I：

10.1161/01.HYP.14.4.453

中图分类号：

R6 [外科学];

学科分类号：

1002 ; 100210 ;

摘要：

引用

页码：453 / 456

页数：4

共 17 条

[1]

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[2]

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DECLERCK, I ;

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[4]

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[5]

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[8] EVIDENCE THAT THE INCREASED CALCIUM SENSITIVITY OF RESISTANCE VESSELS IN SPONTANEOUSLY HYPERTENSIVE RATS IS AN INTRINSIC DEFECT OF THEIR VASCULAR SMOOTH-MUSCLE [J].

MULVANY, MJ ;

KORSGAARD, N ;

NYBORG, N .

CLINICAL AND EXPERIMENTAL HYPERTENSION, 1981, 3 (04) :749-761

[9] NOREPINEPHRINE-INDUCED PHASIC ACTIVITY IN TAIL ARTERIES FROM GENETICALLY HYPERTENSIVE RATS [J].

MYERS, JH ;

LAMB, FS ;

WEBB, RC .

AMERICAN JOURNAL OF PHYSIOLOGY, 1985, 248 (03) :H419-H423

[10] REGULATION OF CALCIUM CURRENT BY INTRACELLULAR CALCIUM IN SMOOTH-MUSCLE CELLS OF RABBIT PORTAL-VEIN [J].

OHYA, Y ;

KITAMURA, K ;

KURIYAMA, H .

CIRCULATION RESEARCH, 1988, 62 (02) :375-383

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