心肌梗死发生和发展过程中的免疫机制研究

被引：15

作者：

王晨阳 ^{[1
]}

刘海燕 ^{[1
]}

曹海涛 ^{[1
]}

曹雪滨 ^{[2
]}

机构：

[1] 解放军医院动物室

[2] 解放军医院全军老年心血管病中心

来源：

解放军医药杂志 | 2014年 / 26卷 / 11期

关键词：

心肌梗死; 心力衰竭; 自身免疫; 白细胞;

D O I：

暂无

中图分类号：

R542.22 [];

学科分类号：

1002 ; 100201 ;

摘要：

<正>在全世界范围内,心血管疾病造成了30%的病死率。冠状动脉粥样硬化性心脏病是心肌梗死和心力衰竭的首要病因。动脉粥样硬化及其并发症不仅仅是简单的脂类失衡,而是环境和遗传的影响导致生物系统和免疫系统的瓦解[1]。在心肌梗死和心力衰竭中几乎牵涉到每种免疫细胞,一些免疫细胞可导致动脉粥样硬化、心肌梗死和心力衰竭,而另一些则可以防止相应的病变。因此免疫细胞在心肌梗死和心力衰竭中的作用研究日益成为热点。

引用

页码：21 / 23

页数：3

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