共 18 条
[1]
PIG3 V, an immortalized human vitiligo melanocyte cell line, expresses dilated endoplasmic reticulum. Le Poole IC,Boissy RE,Sarangarajan R,et al. In Vitro Cellular and Developmental Biology . 2000
[2]
Structural aberration of the rough endoplasmic reticulum and melanosome compart-mentalization in long-term cultures of melanocytes from vitiligo patients. Boissy RE,Liu YY,Medrano EE,et al. Journal of Investigative Dermatology . 1991
[3]
Rab GTPases, intracellular traffic and disease. Seabra MC,Mules EH,Hume AN. Trends in Molecular Medicine . 2002
[4]
The dark side of lysosome-related organelles:specialization of the endocytic pathway for melanosome biogenesis. Raposo G,Marks MS. Traffic . 2002
[5]
Neuron[P]. KUTKOVETSKYI VALENTYN YAKOVYCH;TURTI MARYNA VALENTYNIVNA.UA106289U,2016-04-25
[6]
Quality control and protein folding in the secretory pathway. Trombetta ES,Parodi AJ. Annual Review of Cell and Developmental Biology . 2003
[7]
Chediak-Higashi Syndrome: a rare disorder of lysosomes and lysosome related organelles. Shiflett SL,Kaplan J,Ward DM. Pigment Cell Research . 2002
[8]
Aberrant retention of tyrosinase in the endoplasmic reticulum mediates accelerated degradation of the enzyme and contributes to the dedifferentiated phenotype of amelanotic melanoma cells. Halaban R,Cheng E,Zhang Y,et al. Proceedings of the National Academy of Sciences of the United States of America . 1997
[9]
Proper folding and endoplasmic reticulum to golgi transport of tyrosinase are induced by its substrates, DOPA and tyrosine. Halaban R,Cheng E,Svedine S,et al. Journal of Biological Chemistry . 2001
[10]
Abnorm alacidification of melanom a cells induces tyrosinase retention in the early secretory pathway. Halaban R,Patton RS,Cheng E,etal. Journal of Biochemistry . 2002