糖尿病性骨质疏松发病机制的研究进展

[1] 2型糖尿病与骨质疏松的相关因素分析 [J]. 中国骨质疏松杂志, 2010, 16 (05) : 344 - 346

[2] 老年男性2型糖尿病患者下肢血管病变与骨质疏松的关系 [J]. 中国动脉硬化杂志, 2010, 18 (01) : 75 - 77

[3] 维生素D受体基因多态性与2型糖尿病骨密度的关系(英文) [J]. Chinese Medical Journal, 2001, (11)

[4] Advanced Glycation End Products (AGEs), but not High Glucose, Inhibit the Osteoblastic Differentiation of Mouse Stromal ST2 Cells Through the Suppression of Osterix Expression, and Inhibit Cell Growth and Increasing Cell Apoptosis[J] . Kyoko Okazaki,Toru Yamaguchi,Ken-ichiro Tanaka,Masakazu Notsu,Noriko Ogawa,Shozo Yano,Toshitsugu Sugimoto.Calcified Tissue International . 2012 (4)

[5] High glucose stimulates adipogenic and inhibits osteogenic differentiation in MG-63 cells through cAMP/protein kinase A/extracellular signal-regulated kinase pathway [J]. Molecular and Cellular Biochemistry, 2010, 338 : 115 - 122

[6] Leptin Inhibits Osteoclast Generation[J] . Wayne R.Holloway,Fiona McL.Collier,Cathy J.Aitken,Damian E.Myers,Jason M.Hodge,MaryMalakellis,Tamara J.Gough,Gregory R.Collier,Geoffrey C.Nicholson.J Bone Miner Res . 2009 (2)

[7] Collagen cross-links as a determinant of bone quality: a possible explanation for bone fragility in aging, osteoporosis, and diabetes mellitus[J] . M. Saito,K. Marumo.Osteoporosis International . 2010 (2)

[8] Central regulation of bone mass [J]. SEMINARS IN CELL & DEVELOPMENTAL BIOLOGY, 2008, 19 (05) : 452 - 458

[9] Advanced glycation end products stimulate osteoblast apoptosis via the MAP kinase and cytosolic apoptotic pathways[J] . Mani Alikhani,Zoubin Alikhani,Coy Boyd,Christine M. MacLellan,Markos Raptis,Rongkun Liu,Nicole Pischon,Philip C. Trackman,Louis Gerstenfeld,Dana T. Graves.Bone . 2006 (2)

[10] Transgenic mice with osteoblast-targeted insulin-like growth factor-I show increased bone remodeling [J]. BONE, 2006, 39 (03) : 494 - 504

[1] 2型糖尿病与骨质疏松的相关因素分析 [J]. 中国骨质疏松杂志, 2010, 16 (05) : 344 - 346

[2] 老年男性2型糖尿病患者下肢血管病变与骨质疏松的关系 [J]. 中国动脉硬化杂志, 2010, 18 (01) : 75 - 77

[3] 维生素D受体基因多态性与2型糖尿病骨密度的关系(英文) [J]. Chinese Medical Journal, 2001, (11)

[4] Advanced Glycation End Products (AGEs), but not High Glucose, Inhibit the Osteoblastic Differentiation of Mouse Stromal ST2 Cells Through the Suppression of Osterix Expression, and Inhibit Cell Growth and Increasing Cell Apoptosis[J] . Kyoko Okazaki,Toru Yamaguchi,Ken-ichiro Tanaka,Masakazu Notsu,Noriko Ogawa,Shozo Yano,Toshitsugu Sugimoto.Calcified Tissue International . 2012 (4)

[5] High glucose stimulates adipogenic and inhibits osteogenic differentiation in MG-63 cells through cAMP/protein kinase A/extracellular signal-regulated kinase pathway [J]. Molecular and Cellular Biochemistry, 2010, 338 : 115 - 122

[6] Leptin Inhibits Osteoclast Generation[J] . Wayne R.Holloway,Fiona McL.Collier,Cathy J.Aitken,Damian E.Myers,Jason M.Hodge,MaryMalakellis,Tamara J.Gough,Gregory R.Collier,Geoffrey C.Nicholson.J Bone Miner Res . 2009 (2)

[7] Collagen cross-links as a determinant of bone quality: a possible explanation for bone fragility in aging, osteoporosis, and diabetes mellitus[J] . M. Saito,K. Marumo.Osteoporosis International . 2010 (2)

[8] Central regulation of bone mass [J]. SEMINARS IN CELL & DEVELOPMENTAL BIOLOGY, 2008, 19 (05) : 452 - 458

[9] Advanced glycation end products stimulate osteoblast apoptosis via the MAP kinase and cytosolic apoptotic pathways[J] . Mani Alikhani,Zoubin Alikhani,Coy Boyd,Christine M. MacLellan,Markos Raptis,Rongkun Liu,Nicole Pischon,Philip C. Trackman,Louis Gerstenfeld,Dana T. Graves.Bone . 2006 (2)

[10] Transgenic mice with osteoblast-targeted insulin-like growth factor-I show increased bone remodeling [J]. BONE, 2006, 39 (03) : 494 - 504