植物抗旱的分子机制研究

被引:32
作者
沈元月
黄丛林
张秀海
曹鸣庆
不详
机构
[1] 中国农业大学果树分子发育生物实验室
[2] 北京农业生物技术研究中心
[3] 北京农业生物技术研究中心 北京
[4] 北京
[5] 北京
关键词
干旱; 水分胁迫; 调节蛋白; 功能蛋白; 信号传递;
D O I
暂无
中图分类号
S311 [作物生理学];
学科分类号
090302 ;
摘要
干旱是影响植物生长发育最主要的逆境因子。植物在水分胁迫下会引起一系列分子反应和信号传递,干旱胁迫诱导基因表达一些重要的功能蛋白和调节蛋白以保护细胞不受水分胁迫的伤害。目前已研究证实相关蛋白有跨膜运输蛋白(水通道蛋白、ATP酶等)、水分胁迫调节剂(K+、Na+、蔗糖、脯氨酸、甜菜碱等)、运输或合成相关的酶、Lea蛋白、抗氧化作用相关的酶(SOD、CAT等)、水分胁迫蛋白、调控蛋白(蛋白激酶、转录因子)等。干旱胁迫诱导基因的活化至少涉及4条途径:植物细胞可能通过膨压变化或膜受体的构象变化感知水分胁迫,将胞外信号转为胞内信号,从而触发相应的信号途径,并可导致第二信使(Ca2+、IP3等)生成,在这原始信号被逐级传递放大的过程中,其中2条传递途径是依赖ABA的,另外2条传递途径是不依赖ABA的。通过基因表达调控已分析鉴定出一些水分胁迫有关的顺式作用元件(ABRE、DRE、Myc等)和转录因子(bzip、DREBP、MYC/MYB等)。
引用
收藏
页码:34 / 38
页数:5
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