硫化氢和心肌缺血再灌注损伤

[1] Hydrogen sulfide protects against apoptosis under oxidative stress through SIRT1 pathway in H9c2 cardiomyocytes[J] . Dan Wu,Qingxun Hu,Xinhua Liu,Lilong Pan,Qinghui Xiong,Yi Zhun Zhu.Nitric Oxide . 2014

[2] Involvement of miR-1 in the protective effect of hydrogen sulfide against cardiomyocyte apoptosis induced by ischemia/reperfusion [J]. MOLECULAR BIOLOGY REPORTS, 2014, 41 (10) : 6845 - 6853

[3] Emergence of Hydrogen Sulfide as an Endogenous Gaseous Signaling Molecule in Cardiovascular Disease [J]. CIRCULATION RESEARCH, 2014, 114 (04) : 730 - 737

[4] Passive loss of hydrogen sulfide in biological experiments[J] . Eric R. DeLeon,Gilbrian F. Stoy,Kenneth R. Olson.Analytical Biochemistry . 2011 (1)

[5] Both endogenous and exogenous testosterone decrease myocardial STAT3 activation and SOCS3 expression after acute ischemia and reperfusion [J]. SURGERY, 2009, 146 (02) : 138 - 144

[6] ANTI-APOPTOTIC AND ANTI-INFLAMMATORY EFFECTS OF HYDROGEN SULFIDE IN A RAT MODEL OF REGIONAL MYOCARDIAL I/R [J]. SHOCK, 2009, 31 (03): : 267 - 274

[7] Hydrogen sulfide decreases adenosine triphosphate levels in aortic rings and leads to vasorelaxation via metabolic inhibition[J] . Levente Kiss,Edwin A. Deitch,Csaba Szabó.Life Sciences . 2008 (17)

[8] Effects of hydrogen sulphide on ischaemia–reperfusion injury and ischaemic preconditioning in the isolated, perfused rat heart[J] . Marte Bliks?en,Mari-Liis Kaljusto,Jarle Vaage,K?re-Olav Stensl?kken.European Journal of Cardio-Thoracic Surgery . 2008 (2)

[9] THE PRODUCTION OF HYDROGEN SULFIDE LIMITS MYOCARDIAL ISCHEMIA AND REPERFUSION INJURY AND CONTRIBUTES TO THE CARDIOPROTECTIVE EFFECTS OF PRECONDITIONING WITH ENDOTOXIN, BUT NOT ISCHEMIA IN THE RAT[J] . A. Sivarajah,M.C. McDonald,C. Thiemermann.Shock . 2006 (2)

[10] Role of the JAK–STAT pathway in myocardial injury[J] . Seán P. Barry,Paul A. Townsend,David S. Latchman,Anastasis Stephanou.Trends in Molecular Medicine . 2006 (2)

[1] Hydrogen sulfide protects against apoptosis under oxidative stress through SIRT1 pathway in H9c2 cardiomyocytes[J] . Dan Wu,Qingxun Hu,Xinhua Liu,Lilong Pan,Qinghui Xiong,Yi Zhun Zhu.Nitric Oxide . 2014

[2] Involvement of miR-1 in the protective effect of hydrogen sulfide against cardiomyocyte apoptosis induced by ischemia/reperfusion [J]. MOLECULAR BIOLOGY REPORTS, 2014, 41 (10) : 6845 - 6853

[3] Emergence of Hydrogen Sulfide as an Endogenous Gaseous Signaling Molecule in Cardiovascular Disease [J]. CIRCULATION RESEARCH, 2014, 114 (04) : 730 - 737

[4] Passive loss of hydrogen sulfide in biological experiments[J] . Eric R. DeLeon,Gilbrian F. Stoy,Kenneth R. Olson.Analytical Biochemistry . 2011 (1)

[5] Both endogenous and exogenous testosterone decrease myocardial STAT3 activation and SOCS3 expression after acute ischemia and reperfusion [J]. SURGERY, 2009, 146 (02) : 138 - 144

[6] ANTI-APOPTOTIC AND ANTI-INFLAMMATORY EFFECTS OF HYDROGEN SULFIDE IN A RAT MODEL OF REGIONAL MYOCARDIAL I/R [J]. SHOCK, 2009, 31 (03): : 267 - 274

[7] Hydrogen sulfide decreases adenosine triphosphate levels in aortic rings and leads to vasorelaxation via metabolic inhibition[J] . Levente Kiss,Edwin A. Deitch,Csaba Szabó.Life Sciences . 2008 (17)

[8] Effects of hydrogen sulphide on ischaemia–reperfusion injury and ischaemic preconditioning in the isolated, perfused rat heart[J] . Marte Bliks?en,Mari-Liis Kaljusto,Jarle Vaage,K?re-Olav Stensl?kken.European Journal of Cardio-Thoracic Surgery . 2008 (2)

[9] THE PRODUCTION OF HYDROGEN SULFIDE LIMITS MYOCARDIAL ISCHEMIA AND REPERFUSION INJURY AND CONTRIBUTES TO THE CARDIOPROTECTIVE EFFECTS OF PRECONDITIONING WITH ENDOTOXIN, BUT NOT ISCHEMIA IN THE RAT[J] . A. Sivarajah,M.C. McDonald,C. Thiemermann.Shock . 2006 (2)

[10] Role of the JAK–STAT pathway in myocardial injury[J] . Seán P. Barry,Paul A. Townsend,David S. Latchman,Anastasis Stephanou.Trends in Molecular Medicine . 2006 (2)