共 20 条
[1]
Insulin resistance, hyperlipidemia, and hypertension in mice lacking endothelial nitric oxide synthase. Duplain H,Burcelin R,Sartori C,et al. Circulation . 2001
[2]
Heme is a potent inducer of inflammation in mice and is counteracted by heme oxygenase. Wagener FA,Eggert A,Boerman OC,et al. Blood . 2001
[3]
Identification of protein disulfide isomerase as an endothelial hypoxic stress protein. Graven KK,Molvar C,Roncarati JS,et al. American Journal of Physiology Lung Cellular and Molecular Physiology . 2002
[4]
Domain mapping studies reveal that the M domain of hsp90 serves as a molecular scaffold to regulate AKT-dependent phosphorylation of endothelial nitric oxide synthase and NO release. Fontana J,Fulton D,Chen Y,et al. Circulation Research . 2002
[5]
Nitric oxide synthase gene therapy rapidly reduces adhesion molecule expression and inflammatory cell infiltration in carotid arteries of cholesterol-fed rabbits. Qian H,Neplioueva V,Shetty GA,et al. Circulation . 1999
[6]
HMG-CoA reductase inhibitors: Is the endothelium the main target?. Puddu P,Puddu GM,Muscari A. Cardiology . 2001
[7]
Heat shock protein 90 mediates the balance of nitric oxide and superoxide anion from endothelial nitric-oxide synthase. Pritchard KA,Ackerman AW,Gross ER,et al. Journal of Biological Chemistry . 2001
[8]
Markers of endothelial damage in organ dysfunction and sepsis. Reinhart K,Bayer O,Brunkhorst F,et al. Crit Care Med, 2002, 30(5 Suppl. ) .
[9]
Role of heat shock protein 90 in bradykinin-stimulated endothelial nitric oxide release. Harris MB,Ju H,Venema VJ,et al. General Pharmacology . 2000
[10]
Hsp90 ensures the transition from the early Ca2+-dependent to the late phosphorylation-dependent activation of the endothelial nitric-oxide synthase in vascular endothelial growth factor-exposed endothelial cells. Brouet A,Sonveaux P,Dessy C,et al. Journal of Biological Chemistry . 2001