单核因子与血清阴性脊柱关节病

[1] Serum cytokines(IL-6, TNF,IL-1, IFN-γ)in ankylosing spondylitis. Gratacos J,Collodo A,Filella X,et al. British Journal of Rheumatology . 1994

[2] Patterns of expression of tumor necrosis factorα, tumor necrosis factorβ, and other receptors in synovia of patients with juvenile rheumatoid arthritis and juvenile spondylarthropathy. Grom AA,Murry KJ,Luyrink L,et al. Arthritis and Rheumatism . 1996

[3] Use of immunohistologic and in situ hybridization techniques the examination of sacroiliac joint biopsy specimens from patients from patients with ankylosing spondylitis. Braun J,Bollow M,Neure L,et al. Arthritis and Rheumatism . 1995

[4] Sulfasalazine inhibition of endotoxin induced interleukin-1 production from human monocytes in vitro. Remving L. Scandinavian Journal of Immunology . 1988

[5] Monocyte activity in Behcet′s disease. Sahin S,Lawrence R,Direskeneki H,et al. British Journal of Rheumatology . 1996

[6] Tumour necrosis factor in synovial exudates. Giovine FS,Nuki G,Duff GW,et al. Annals of the Rheumatic Diseases . 1988

[7] Production of tumour necrosis factor and interleukin 1 by monocytes of patients with previous Yersinia arthritis. Repo H,J ttel M,Leirisalo-Repo M,et al. Clinical and Experimental Immunology . 1988

[8] Interleukin-6, acute phase reactants and clinical status in ankylosing spondylitis. Bilgic A,Tutuncu ZN,Kennedy LG,et al. British Journal of Rheumatology . 1993

[9] Thalidomide selectively inhibits tumor necrosis factor α production by stimulated human monocytes. Elizabeth P,Euzenir N,Ruth G,et al. The Journal of Experimental Medicine . 1991

[10] Recombinant human tumor necrosis factors alpha and bets stimulate fibroblasts to produce hemopoietic growth factors in vitro. Zucali JR,Broxmeyer HE,Cross MA,et al. J Immunol . 1988

[1] Serum cytokines(IL-6, TNF,IL-1, IFN-γ)in ankylosing spondylitis. Gratacos J,Collodo A,Filella X,et al. British Journal of Rheumatology . 1994

[2] Patterns of expression of tumor necrosis factorα, tumor necrosis factorβ, and other receptors in synovia of patients with juvenile rheumatoid arthritis and juvenile spondylarthropathy. Grom AA,Murry KJ,Luyrink L,et al. Arthritis and Rheumatism . 1996

[3] Use of immunohistologic and in situ hybridization techniques the examination of sacroiliac joint biopsy specimens from patients from patients with ankylosing spondylitis. Braun J,Bollow M,Neure L,et al. Arthritis and Rheumatism . 1995

[4] Sulfasalazine inhibition of endotoxin induced interleukin-1 production from human monocytes in vitro. Remving L. Scandinavian Journal of Immunology . 1988

[5] Monocyte activity in Behcet′s disease. Sahin S,Lawrence R,Direskeneki H,et al. British Journal of Rheumatology . 1996

[6] Tumour necrosis factor in synovial exudates. Giovine FS,Nuki G,Duff GW,et al. Annals of the Rheumatic Diseases . 1988

[7] Production of tumour necrosis factor and interleukin 1 by monocytes of patients with previous Yersinia arthritis. Repo H,J ttel M,Leirisalo-Repo M,et al. Clinical and Experimental Immunology . 1988

[8] Interleukin-6, acute phase reactants and clinical status in ankylosing spondylitis. Bilgic A,Tutuncu ZN,Kennedy LG,et al. British Journal of Rheumatology . 1993

[9] Thalidomide selectively inhibits tumor necrosis factor α production by stimulated human monocytes. Elizabeth P,Euzenir N,Ruth G,et al. The Journal of Experimental Medicine . 1991

[10] Recombinant human tumor necrosis factors alpha and bets stimulate fibroblasts to produce hemopoietic growth factors in vitro. Zucali JR,Broxmeyer HE,Cross MA,et al. J Immunol . 1988