冠状动脉支架置入后血栓形成的原因和机制

被引:4
作者
庞兴学 [1 ]
王显 [2 ]
机构
[1] 北京老年医院心内科
[2] 不详
关键词
支架内血栓形成; DES; 内皮细胞; 冠状动脉; 支架血栓; 内皮化; 支架置入; BMS; 分叉病变; 血小板粘附; 再狭窄率; 复杂病变; 支架内再狭窄;
D O I
暂无
中图分类号
R541.4 [冠状动脉(粥样)硬化性心脏病(冠心病)];
学科分类号
摘要
<正>自1987年支架置入术应用于经皮冠状动脉介入治疗(percutanenous coronary intervention,PCI)领域以来,成功解决了单纯PTCA术的急性血管弹性回缩导致再狭窄的问题,但金属裸支架(bare-metal stents,BMS)仍然有约30%的再狭窄率,而药物洗脱支架,特别是雷帕霉素和紫杉醇药物支架的出现,将再狭窄率进一步降至10%以内,随后关于DES与支架内血栓的临床研究显示:雷帕霉素和紫杉醇作
引用
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页码:432 / 434
页数:3
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