少突胶质细胞与缺血性脑损伤

[1] E ffects ofmatrix metalloproteinase-9 gene knock-out on the proteolysis of blood-brain barrier and white matter components after cerebral ischem ia. AsahiM,W ang X,Mori T,et al. The Journal of Neuroscience . 2001

[2] Ampa /kainate receptor activation mediates hypoxic oligodendrocyte death and axonal injury in cerebral white matter. Tekkok SB,GoldbergMP. The Journal of Neuroscience . 2001

[3] The oligodendroglia cytoskeleton in health and disease. R ichter-Landsberg C. Journal of Neuroscience Research . 2000

[4] Contribution of m icroglia /macrophages to expansion of infarction and response of oligodendrocytes after focal cerebral ischem ia in rats. Mabuchi T,K itagawa K,Ohtsuki T,et al. Stroke . 2000

[5] AMPA receptor-mediated toxicity in oligodendrocyte progenitors involves free radical generation and activation of JNK, calpain and caspase 3. LiuHN,G iasson B I,MushynskiWE,et al. Journal of Neurochemistry . 2002

[6] Caspases determ ine the vulnerability of oligodendrocytes in the ischem ic brain. ShibataM,H isahara S,HaraH,et al. The Journal of Clinical Investigation . 2000

[7] D issociative increase of oligodendrocyte progenitor cells between young and aged rats after transient cerebral ischem ia. Ohta K,IwaiM,SatoK,et al. Neuroscience Letters . 2003

[8] Caspase-dependent and caspase-independent oligodendrocyte death mediated by AMPA and kainate receptors. Sanchez-Gomez MV,A lberdi E,Ibarretxe G,et al. The Journal of Neuroscience . 2003

[9] Non-N-methyl-D-aspartate glutamate receptors mediate oxygen-glucose deprivation-induced oligodendroglial injury. Yoshioka A,Yamaya Y,Saiki S,et al. Brain Research . 2000

[10] Activation ofNG2-positive oligodendrocyte progenitor cells during post-ischem ic reperfusion in the rat brain. Tanaka K,Nogawa S,Ito D,et al. NeuroReport . 2001

[1] E ffects ofmatrix metalloproteinase-9 gene knock-out on the proteolysis of blood-brain barrier and white matter components after cerebral ischem ia. AsahiM,W ang X,Mori T,et al. The Journal of Neuroscience . 2001

[2] Ampa /kainate receptor activation mediates hypoxic oligodendrocyte death and axonal injury in cerebral white matter. Tekkok SB,GoldbergMP. The Journal of Neuroscience . 2001

[3] The oligodendroglia cytoskeleton in health and disease. R ichter-Landsberg C. Journal of Neuroscience Research . 2000

[4] Contribution of m icroglia /macrophages to expansion of infarction and response of oligodendrocytes after focal cerebral ischem ia in rats. Mabuchi T,K itagawa K,Ohtsuki T,et al. Stroke . 2000

[5] AMPA receptor-mediated toxicity in oligodendrocyte progenitors involves free radical generation and activation of JNK, calpain and caspase 3. LiuHN,G iasson B I,MushynskiWE,et al. Journal of Neurochemistry . 2002

[6] Caspases determ ine the vulnerability of oligodendrocytes in the ischem ic brain. ShibataM,H isahara S,HaraH,et al. The Journal of Clinical Investigation . 2000

[7] D issociative increase of oligodendrocyte progenitor cells between young and aged rats after transient cerebral ischem ia. Ohta K,IwaiM,SatoK,et al. Neuroscience Letters . 2003

[8] Caspase-dependent and caspase-independent oligodendrocyte death mediated by AMPA and kainate receptors. Sanchez-Gomez MV,A lberdi E,Ibarretxe G,et al. The Journal of Neuroscience . 2003

[9] Non-N-methyl-D-aspartate glutamate receptors mediate oxygen-glucose deprivation-induced oligodendroglial injury. Yoshioka A,Yamaya Y,Saiki S,et al. Brain Research . 2000

[10] Activation ofNG2-positive oligodendrocyte progenitor cells during post-ischem ic reperfusion in the rat brain. Tanaka K,Nogawa S,Ito D,et al. NeuroReport . 2001