一氧化氮在脑缺血预处理中的作用及其机制

[1] Delayed neural damage is induced by iNOS-expressing microglia in a brain injury model [J]. NEUROSCIENCE LETTERS, 2010, 473 (02) : 146 - 150

[2] What is the real physiological NO concentration in vivo? [J]. NITRIC OXIDE-BIOLOGY AND CHEMISTRY, 2009, 21 (02): : 92 - 103

[3] Neuronal nitric oxide synthase: Structure, subcellular localization, regulation, and clinical implications[J] . Li Zhou,Dong-Ya Zhu.Nitric Oxide . 2009 (4)

[4] Preconditioning neuroprotection in global cerebral ischemia involves NMDA receptor-mediated ERK-JNK3 crosstalk [J]. NEUROSCIENCE RESEARCH, 2009, 63 (03) : 205 - 212

[5] A dual role of the NF-κB pathway in neonatal hypoxic-ischemic brain damage [J]. STROKE, 2008, 39 (09) : 2578 - 2586

[6] Endogenous neuroprotection: Mitochondria as gateways to cerebral preconditioning?[J] . Ulrich Dirnagl,Andreas Meisel.Neuropharmacology . 2008 (3)

[7] The Protective Effect of Ischemic Postconditioning Against Ischemic Injury: From the Heart to the Brain[J] . Heng Zhao.Journal of Neuroimmune Pharmacology . 2007 (4)

[8] Preconditioning Reprograms the Response to Ischemic Injury and Primes the Emergence of Unique Endogenous Neuroprotective Phenotypes: A Speculative Synthesis[J] . Mary P. Stenzel-Poore,Susan L. Stevens,Jeffrey S. King,Roger P. Simon.Stroke . 2007 (2, P)

[9] Vascular response to hypoxic preconditioning in the immature brain [J]. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM, 2007, 27 (05) : 928 - 938

[10] The Mitochondrial K＜sub＞ATP＜/sub＞ Channel Opener BMS-191095 Reduces Neuronal Damage after Transient Focal Cerebral Ischemia in Rats[J] . Keita Mayanagi,Tamás Gáspár,Prasad VG Katakam,Béla Kis,David W Busija.Journal of Cerebral Blood Flow ＆ Metabolism . 2007 (2)

[1] Delayed neural damage is induced by iNOS-expressing microglia in a brain injury model [J]. NEUROSCIENCE LETTERS, 2010, 473 (02) : 146 - 150

[2] What is the real physiological NO concentration in vivo? [J]. NITRIC OXIDE-BIOLOGY AND CHEMISTRY, 2009, 21 (02): : 92 - 103

[3] Neuronal nitric oxide synthase: Structure, subcellular localization, regulation, and clinical implications[J] . Li Zhou,Dong-Ya Zhu.Nitric Oxide . 2009 (4)

[4] Preconditioning neuroprotection in global cerebral ischemia involves NMDA receptor-mediated ERK-JNK3 crosstalk [J]. NEUROSCIENCE RESEARCH, 2009, 63 (03) : 205 - 212

[5] A dual role of the NF-κB pathway in neonatal hypoxic-ischemic brain damage [J]. STROKE, 2008, 39 (09) : 2578 - 2586

[6] Endogenous neuroprotection: Mitochondria as gateways to cerebral preconditioning?[J] . Ulrich Dirnagl,Andreas Meisel.Neuropharmacology . 2008 (3)

[7] The Protective Effect of Ischemic Postconditioning Against Ischemic Injury: From the Heart to the Brain[J] . Heng Zhao.Journal of Neuroimmune Pharmacology . 2007 (4)

[8] Preconditioning Reprograms the Response to Ischemic Injury and Primes the Emergence of Unique Endogenous Neuroprotective Phenotypes: A Speculative Synthesis[J] . Mary P. Stenzel-Poore,Susan L. Stevens,Jeffrey S. King,Roger P. Simon.Stroke . 2007 (2, P)

[9] Vascular response to hypoxic preconditioning in the immature brain [J]. JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM, 2007, 27 (05) : 928 - 938

[10] The Mitochondrial K＜sub＞ATP＜/sub＞ Channel Opener BMS-191095 Reduces Neuronal Damage after Transient Focal Cerebral Ischemia in Rats[J] . Keita Mayanagi,Tamás Gáspár,Prasad VG Katakam,Béla Kis,David W Busija.Journal of Cerebral Blood Flow ＆ Metabolism . 2007 (2)