基质金属蛋白酶与充血性心力衰竭时心脏重构

被引:12
作者
沈文俊
马端
机构
[1] 复旦大学上海医学院分子医学教育部重点实验室
关键词
心脏重构; 心力衰竭,充血性; 基质金属蛋白酶;
D O I
暂无
中图分类号
R541.6 [血液循环衰竭];
学科分类号
摘要
引用
收藏
页码:379 / 381
页数:3
相关论文
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[1]  
The matrix metalloproteinase (MMP) and tissue inhibitor of metalloproteinase (TIMP) genes. Vincenti M P. Methods in Molecular Biology . 2001
[2]  
Selective matrix metalloproteinase inhibition with developing heart failure: effects on left ventricular function and structure. King M K,Coker M L,James A G,et al. Circulation Research . 2003
[3]  
Glioma cell extracellular matrix metalloproteinases inducer (EMMPRIN) (CD47) stimulates production of membrane-type matrix metalloproteinases and activated gelatinase A in cocultures with brain-derived fibroblasts. Sameshima T,Nabeshima K,Tpple B P,et al. Cancer Letters . 2000
[4]  
Introduction to the Matrix Metalloproteinases ( MMPs ). Woessner J F Jr,Nagase H. In: Matrix Metalloproteinases and TIMPs . 2000
[5]  
Downregulation of matrix metalloproteinases and reduction in collagen damage in the failing human heart after support with left ventricular assist devices. Li Y Y,Feng Y Q,McTiernan C F,et al. Circulation . 2001
[6]  
Effects of gene deletion of the tissue inhibitor of the matrix metalloproteinasetype 1 (TIMP-1) on left ventricular geometry and function in mice. Roten L,Nemoto S,Simsic J,et al. Journal of Molecular and Cellular Cardiology . 2000
[7]  
Human membrane type-2 matrix metalloproteinase is defective in cellassociated activation of progelatinase A. Miyamori H,Takino T,Seiki M,et al. Biochemical and Biophysical Research Communications . 2000
[8]  
Increased matrix metalloproteinase activity and selective upregulation in LV myocardium from patients with end-stage dilated cardiomyopathy. Thomas C V,Coker M L,Zellner J L,et al. Circulation . 1998
[9]  
Matrix metalloproteinase inhibition during the development of congestive heart failure: effects on left ventricular dimensions and function. Spinale F G,Coker M L,Thomas C V,et al. Circulation Research . 1999
[10]  
Matrix metalloproteinase inhibition attenuates left ventricular remodeling and dysfunction in a rat model of progrssive heart failure. Peterson J T,Hallak H,Johnson L,et al. Circulation . 2001