要重视卒中后痴呆的研究

[1] Apolipoprotein E: a pathological chaperone protein in patients with cerebral and systemic amyloid. Wisniewski T,Frangione B. Neuroscience Letters . 1992

[2] Presenilin-1 mutation increases neuronal vulnerability to focal ischemia in vivo and to hypoxia and glucose deprivation in cell culture: involvement of perturbed calcium homeostasis. Mattson MP,Zhu HY,Yu J,et al. The Journal of Neuroscience . 2000

[3] Binswanger encephalopathy: a review. Fisher CM. Journal of Neurology . 1989

[4] Clinical and radiological determinants of prestroke cognitive decline in a stroke cohort. Pohjasvaara T,Mantyla R,Aronen HJ,et al. Journal of Neurology Neurosurgery and Psychiatry . 1999

[5] A quantitative MRI study of vascular dementia. Liu CK,Miller BL,Cummings JL,et al. Neurology . 1992

[6] Alzheimer disease as a vascular disorder:nosological evidence. de la Torre JC. Stroke . 2002

[7] Relative frequencies of Alzheimer disease, Lewy body, vascular and frontotemporal dementia, and hippocampal sclerosis in the State of Florida Brain Bank. Barker W W,Luis C A,Kashuba A,et al. Alzheimer Disease and Associated Disorders . 2002

[8] Clinicopathological analysis of dementia disorders in the elderly. Jellinger K,Danielczyk W,Fischer P,et al. Journal of the Neurological Sciences . 1990

[9] Absence of vascular dementia in an autopsy series from a dementia clinic. Nolan KA,Lino MM,Seligmann AW,et al. Journal of the American Geriatrics Society . 1998

[10] Blood brain barrier transport of circulating Alzheimer′s amyloid beta. Zlokovic BV,Ghiso J,Mackic JB. Biochemical and Biophysical Research Communications . 1993

[1] Apolipoprotein E: a pathological chaperone protein in patients with cerebral and systemic amyloid. Wisniewski T,Frangione B. Neuroscience Letters . 1992

[2] Presenilin-1 mutation increases neuronal vulnerability to focal ischemia in vivo and to hypoxia and glucose deprivation in cell culture: involvement of perturbed calcium homeostasis. Mattson MP,Zhu HY,Yu J,et al. The Journal of Neuroscience . 2000

[3] Binswanger encephalopathy: a review. Fisher CM. Journal of Neurology . 1989

[4] Clinical and radiological determinants of prestroke cognitive decline in a stroke cohort. Pohjasvaara T,Mantyla R,Aronen HJ,et al. Journal of Neurology Neurosurgery and Psychiatry . 1999

[5] A quantitative MRI study of vascular dementia. Liu CK,Miller BL,Cummings JL,et al. Neurology . 1992

[6] Alzheimer disease as a vascular disorder:nosological evidence. de la Torre JC. Stroke . 2002

[7] Relative frequencies of Alzheimer disease, Lewy body, vascular and frontotemporal dementia, and hippocampal sclerosis in the State of Florida Brain Bank. Barker W W,Luis C A,Kashuba A,et al. Alzheimer Disease and Associated Disorders . 2002

[8] Clinicopathological analysis of dementia disorders in the elderly. Jellinger K,Danielczyk W,Fischer P,et al. Journal of the Neurological Sciences . 1990

[9] Absence of vascular dementia in an autopsy series from a dementia clinic. Nolan KA,Lino MM,Seligmann AW,et al. Journal of the American Geriatrics Society . 1998

[10] Blood brain barrier transport of circulating Alzheimer′s amyloid beta. Zlokovic BV,Ghiso J,Mackic JB. Biochemical and Biophysical Research Communications . 1993