脑缺血再灌注损伤中细胞凋亡的研究进展

被引:58
作者
鲍磊
张铮
机构
[1] 南京医科大学附属南京第一医院
关键词
D O I
暂无
中图分类号
R743 [脑血管疾病];
学科分类号
100204 [神经病学];
摘要
<正>近年来,脑血管病的发病率逐年升高,在这一疾病和相应的治疗过程中,脑缺血/再灌注损伤(ischemia reperfusion injury)的病理生理过程中起着重要的作
引用
收藏
页码:487 / 489
页数:3
相关论文
共 9 条
[1]
A dual role of the NF-κB pathway in neonatal hypoxic-ischemic brain damage [J].
Nijboer, Cora H. ;
Heijnen, Cobi J. ;
Groenendaal, Floris ;
May, Michael J. ;
van Bel, Frank ;
Kavelaars, Annemieke .
STROKE, 2008, 39 (09) :2578-2586
[2]
Effects of ATP sensitive potassium channel opener on the mRNA and protein expressions of caspase-12 after cerebral ischemia-reperfusion in rats [J].
Zhang H. ;
Song L.-C. ;
Jia C.-H. ;
Lu Y.-L. .
Neuroscience Bulletin, 2008, 24 (1) :7-12
[3]
A novel inhibitor of advanced glycation and endoplasmic reticulum stress reduces infarct volume in rat focal cerebral ischemia.[J].Shunya Takizawa;Yuko Izuhara;Yasuko Kitao;Osamu Hori;Satoshi Ogawa;Yuko Morita;Shigeharu Takagi;Charles van Ypersele de Strihou;Toshio Miyata.Brain Research.2007,
[4]
Pinacidil reduces neuronal apoptosis following cerebral ischemia-reperfusion in rats through both mitochondrial and death-receptor signal pathways [J].
Zhang H. ;
Song L.-C. ;
Liu Y.-Y. ;
Ma Y. ;
Lu Y.-L. .
Neuroscience Bulletin, 2007, 23 (3) :145-150
[5]
Nuclear and mitochondrial conversations in cell death: PARP-1 and AIF signaling [J].
Hong, SJ ;
Dawson, TM ;
Dawson, VL .
TRENDS IN PHARMACOLOGICAL SCIENCES, 2004, 25 (05) :259-264
[6]
Induction of GRP78 by ischemic preconditioning reduces endoplasmic reticulum stress and prevents delayed neuronal cell death [J].
Hayashi, T ;
Saito, A ;
Okuno, S ;
Ferrand-Drake, M ;
Chan, PH .
JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM, 2003, 23 (08) :949-961
[7]
Activation of caspase-12 by endoplasmic reticulum stress induced by transient middle cerebral artery occlusion in mice [J].
Shibata, M ;
Hattori, H ;
Sasaki, T ;
Gotoh, J ;
Hamada, J ;
Fukuuchi, Y .
NEUROSCIENCE, 2003, 118 (02) :491-499
[8]
The unfolding tale of the unfolded protein response [J].
Ma, YJ ;
Hendershot, LM .
CELL, 2001, 107 (07) :827-830
[9]
双苯氟嗪抗大鼠全脑缺血再灌注所致海马CA1区神经元损伤的作用机制(英文).[J].张英俊;郭扬;贾庆中;王永利;张海林.药学学报.2005, 02