脓毒症心肌抑制研究进展

被引：28

作者：

杨乐 ^{[1
]}

邹晓静 ^{[2
]}

李树生 ^{[1
]}

万磊 ^{[1
]}

姚尚龙 ^{[2
]}

杨光田 ^{[1
]}

机构：

[1] 华中科技大学同济医学院附属同济医院

[2] 华中科技大学同济医学院附属协和医院

来源：

内科急危重症杂志 | 2010年 / 16卷 / 01期

关键词：

脓毒症; 心肌抑制; 研究; 进展;

D O I：

暂无

中图分类号：

R459.7 [急症、急救处理];

学科分类号：

100231 [临床病理学];

摘要：

引用

收藏

页码：46 / 48

页数：3

相关论文

共 9 条

[1]

感染性休克治疗新进展 [J].

常锦雄 ;

温端改 .

内科急危重症杂志, 1999, (03) :130-131

[2]

Transcutaneous vagus nerve stimulation reduces serum high mobility group box 1 levels and improves survival in murine sepsis.[J].Jared M. Huston;Margot Gallowitsch-Puerta;Mahendar Ochani;Kanta Ochani;Renqi Yuan;Mauricio Rosas-Ballina;Mala Ashok;Richard S. Goldstein;Sangeeta Chavan;Valentin A. Pavlov;Christine N. Metz;Huan Yang;Christopher J. Czura;Haichao Wang;Kevin J. Tracey.Critical Care Medicine.2007, 12

[3]

Mechanisms of sepsis-induced cardiac dysfunction [J].

Rudiger, Alain ;

Singer, Mervyn .

CRITICAL CARE MEDICINE, 2007, 35 (06) :1599-1608

[4]

Endotoxin-induced heart dysfunction in rats: Assessment of myocardial perfusion and permeability and the role of fluid resuscitation [J].

Chagnon, F ;

Bentourkia, M ;

Lecomte, R ;

Lessard, M ;

Lesur, O .

CRITICAL CARE MEDICINE, 2006, 34 (01) :127-133

[5]

Lysozyme binding to endocardial endothelium mediates myocardial depression by the nitric oxide guanosine 3′,5′ monophosphate pathway in sepsis [J].

Mink, SN ;

Bose, R ;

Roberts, DE ;

Jacobs, H ;

Duke, K ;

Bose, D ;

Cheng, ZQ ;

Light, RB .

JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY, 2005, 39 (04) :615-625

[6]

Tumor necrosis factor-α-induced caspase activation mediates endotoxin-related cardiac dysfunction [J].

Carlson, DL ;

Willis, MS ;

White, J ;

Horton, JW ;

Giroir, BP .

CRITICAL CARE MEDICINE, 2005, 33 (05) :1021-1028

[7]

Ventricular myocyte caspases are directly responsible for endotoxin-induced cardiac dysfunction [J].

Lancel, S ;

Joulin, O ;

Favory, R ;

Goossens, JF ;

Kluza, J ;

Chopin, C ;

Formstecher, P ;

Marchetti, P ;

Neviere, R .

CIRCULATION, 2005, 111 (20) :2596-2604

[8]

Mitochondrial dysfunction in septic shock and multiple organ dysfunction syndrome.[J].Elliott D. Crouser.Mitochondrion.2004, 5

[9]

G protein and adenylate cyclase complex-mediated signal transduction in the rat heart during sepsis [J].

Wu, LL ;

Yang, SL ;

Yang, RC ;

Hsu, HK ;

Hsu, C ;

Dong, LW ;

Liu, MS .

SHOCK, 2003, 19 (06) :533-537