线粒体与缺氧缺血性脑细胞凋亡

[1] BAX Contributes to Apoptotic-Like Death Following Neonatal Hypoxia-Ischemia: Evidence for Distinct Apoptosis Pathways[J] . Margaret E. Gibson,Byung Hee Han,Junjeong Choi,C. Michael Knudson,Stanley Korsmeyer,Maia Parsadanian,David M. Holtzman.Molecular Medicine . 2001 (9)

[2] Identification of DIABLO, a Mammalian Protein that Promotes Apoptosis by Binding to and Antagonizing IAP Proteins[J] . Anne M Verhagen,Paul G Ekert,Miha Pakusch,John Silke,Lisa M Connolly,Gavin E Reid,Robert L Moritz,Richard J Simpson,David L Vaux.Cell . 2000 (1)

[3] Impairment of mitochondrial respiration after cerebral hypoxia–ischemia in immature rats: relationship to activation of caspase-3 and neuronal injury[J] . Malgorzata Puka-Sundvall,Camilla Wallin,Eric Gilland,Ulrika Hallin,Xiaoyang Wang,Mats Sandberg,Jan-Olof Karlsson,Klas Blomgren,Henrik Hagberg.Developmental Brain Research . 2000 (1)

[4] Smac, a Mitochondrial Protein that Promotes Cytochrome c–Dependent Caspase Activation by Eliminating IAP Inhibition[J] . Chunying Du,Min Fang,Yucheng Li,Lily Li,Xiaodong Wang.Cell . 2000 (1)

[5] Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis [J]. CELL, 1998, 94 (04) : 491 - 501

[6] Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade [J]. CELL, 1997, 91 (04) : 479 - 489

[7] Control of apoptosis by the cellular ATP level [J]. FEBS LETTERS, 1996, 378 (02) : 107 - 110

[8] Glutamate-induced neuronal death: A succession of necrosis or apoptosis depending on mitochondrial function[J] . Maria Ankarcrona,Jeannette M. Dypbukt,Emanuela Bonfoco,Boris Zhivotovsky,Sten Orrenius,Stuart A. Lipton,Pierluigi Nicotera.Neuron . 1995 (4)

[1] BAX Contributes to Apoptotic-Like Death Following Neonatal Hypoxia-Ischemia: Evidence for Distinct Apoptosis Pathways[J] . Margaret E. Gibson,Byung Hee Han,Junjeong Choi,C. Michael Knudson,Stanley Korsmeyer,Maia Parsadanian,David M. Holtzman.Molecular Medicine . 2001 (9)

[2] Identification of DIABLO, a Mammalian Protein that Promotes Apoptosis by Binding to and Antagonizing IAP Proteins[J] . Anne M Verhagen,Paul G Ekert,Miha Pakusch,John Silke,Lisa M Connolly,Gavin E Reid,Robert L Moritz,Richard J Simpson,David L Vaux.Cell . 2000 (1)

[3] Impairment of mitochondrial respiration after cerebral hypoxia–ischemia in immature rats: relationship to activation of caspase-3 and neuronal injury[J] . Malgorzata Puka-Sundvall,Camilla Wallin,Eric Gilland,Ulrika Hallin,Xiaoyang Wang,Mats Sandberg,Jan-Olof Karlsson,Klas Blomgren,Henrik Hagberg.Developmental Brain Research . 2000 (1)

[4] Smac, a Mitochondrial Protein that Promotes Cytochrome c–Dependent Caspase Activation by Eliminating IAP Inhibition[J] . Chunying Du,Min Fang,Yucheng Li,Lily Li,Xiaodong Wang.Cell . 2000 (1)

[5] Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis [J]. CELL, 1998, 94 (04) : 491 - 501

[6] Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade [J]. CELL, 1997, 91 (04) : 479 - 489

[7] Control of apoptosis by the cellular ATP level [J]. FEBS LETTERS, 1996, 378 (02) : 107 - 110

[8] Glutamate-induced neuronal death: A succession of necrosis or apoptosis depending on mitochondrial function[J] . Maria Ankarcrona,Jeannette M. Dypbukt,Emanuela Bonfoco,Boris Zhivotovsky,Sten Orrenius,Stuart A. Lipton,Pierluigi Nicotera.Neuron . 1995 (4)