骨关节炎软骨退变的相关性研究

[1] 雌激素受体等位基因多态性与骨关节炎的关系 [J]. 中华风湿病学杂志, 2004, (10) : 583 - 586

[2] 遗传因素和机械因素与膝骨性关节炎发病年龄的关系 [J]. 中华医学遗传学杂志, 2010, (06)

[3] Sex hormones and structural changes in osteoarthritis: A systematic review[J] . Stephanie K. Tanamas,Pushpika Wijethilake,Anita E. Wluka,Miranda L. Davies-Tuck,Donna M. Urquhart,Yuanyuan Wang,Flavia M. Cicuttini.Maturitas . 2011 (2)

[4] The infrapatellar fat pad should be considered as an active osteoarthritic joint tissue: a narrative review[J] . S. Clockaerts,Y.M. Bastiaansen-Jenniskens,J. Runhaar,G.J.V.M. Van Osch,J.F. Van Offel,J.A.N. Verhaar,L.S. De Clerck,J. Somville.Osteoarthritis and Cartilage . 2010 (7)

[5] Human cartilage glycoprotein 39-biomarker of joint damage in knee osteoarthritis [J]. INTERNATIONAL ORTHOPAEDICS, 2009, 33 (04) : 1165 - 1170

[6] Chondrocyte hypertrophy can be induced by a cryptic sequence of type II collagen and is accompanied by the induction of MMP-13 and collagenase activity: Implications for development and arthritis[J] . Elena V. Tchetina,Masahiko Kobayashi,Tadashi Yasuda,Tineke Meijers,Isabelle Pidoux,A. Robin Poole.Matrix Biology . 2007 (4)

[7] Expression of proteinase-activated receptors (PAR)-2 in articular chondrocytes is modulated by IL-1β, TNF-α and TGF-β[J] . Y. Xiang,K. Masuko-Hongo,T. Sekine,H. Nakamura,K. Yudoh,K. Nishioka,T. Kato.Osteoarthritis and Cartilage . 2006 (11)

[8] Mitochondrial activity is modulated by TNFα and IL-1β in normal human chondrocyte cells [J]. OSTEOARTHRITIS AND CARTILAGE, 2006, 14 (10) : 1011 - 1022

[9] Metabolic Mechanisms in the Pathogenesis of Osteoarthritis – A Review[J] . Bruce Burnett,Robert Levy,Brian Cole.J Knee Surg . 2006 (03)

[10] Pathogenesis and management of pain in osteoarthritis [J]. LANCET, 2005, 365 (9463) : 965 - 973

[1] 雌激素受体等位基因多态性与骨关节炎的关系 [J]. 中华风湿病学杂志, 2004, (10) : 583 - 586

[2] 遗传因素和机械因素与膝骨性关节炎发病年龄的关系 [J]. 中华医学遗传学杂志, 2010, (06)

[3] Sex hormones and structural changes in osteoarthritis: A systematic review[J] . Stephanie K. Tanamas,Pushpika Wijethilake,Anita E. Wluka,Miranda L. Davies-Tuck,Donna M. Urquhart,Yuanyuan Wang,Flavia M. Cicuttini.Maturitas . 2011 (2)

[4] The infrapatellar fat pad should be considered as an active osteoarthritic joint tissue: a narrative review[J] . S. Clockaerts,Y.M. Bastiaansen-Jenniskens,J. Runhaar,G.J.V.M. Van Osch,J.F. Van Offel,J.A.N. Verhaar,L.S. De Clerck,J. Somville.Osteoarthritis and Cartilage . 2010 (7)

[5] Human cartilage glycoprotein 39-biomarker of joint damage in knee osteoarthritis [J]. INTERNATIONAL ORTHOPAEDICS, 2009, 33 (04) : 1165 - 1170

[6] Chondrocyte hypertrophy can be induced by a cryptic sequence of type II collagen and is accompanied by the induction of MMP-13 and collagenase activity: Implications for development and arthritis[J] . Elena V. Tchetina,Masahiko Kobayashi,Tadashi Yasuda,Tineke Meijers,Isabelle Pidoux,A. Robin Poole.Matrix Biology . 2007 (4)

[7] Expression of proteinase-activated receptors (PAR)-2 in articular chondrocytes is modulated by IL-1β, TNF-α and TGF-β[J] . Y. Xiang,K. Masuko-Hongo,T. Sekine,H. Nakamura,K. Yudoh,K. Nishioka,T. Kato.Osteoarthritis and Cartilage . 2006 (11)

[8] Mitochondrial activity is modulated by TNFα and IL-1β in normal human chondrocyte cells [J]. OSTEOARTHRITIS AND CARTILAGE, 2006, 14 (10) : 1011 - 1022

[9] Metabolic Mechanisms in the Pathogenesis of Osteoarthritis – A Review[J] . Bruce Burnett,Robert Levy,Brian Cole.J Knee Surg . 2006 (03)

[10] Pathogenesis and management of pain in osteoarthritis [J]. LANCET, 2005, 365 (9463) : 965 - 973