碱性成纤维细胞生长因子治疗脑梗死研究进展

Endogenous neuroprotection factors and traumatic brain injury :mechanisms of action and implications for therapy. Mattson M P,Scheff S W. Journal of Neurotrauma . 1994

Basic F G Fregulates the expression of a functional 71k D N M D Areceptor protein that mediates calcium influx and neurotoxicity in hippocampal neurons. Mattson M P,Kumar K N,Wang H,et al. Journal of the Neurological Sciences . 1993

Hypotensive activity offibroblast growth factor. Cuevas P,Carceller F,Ortega S,et al. Science . 1991

Intracisternal basic fibroblast growth factor enhances functional recovery and upregulates theexpression of a molecular marker of neuronal sprouting following focalcerebral infarction. Kawamata T,Dietrich W D,Schallert T,et al. Proc Natl Acad Sci U S A . 1997

Delayed intravenous administration of basic fibroblast growth factor reduces infarct volume in a model offocal cerebral ischemia/reperfusion in the rat. Jiang N,Finklestein S P,Do T,et al. Journal of the Neurological Sciences . 1996

Usedependent growth of pyramidal neurons afterneocortical damage. Jones T A,Schallert T. Journal of the Neurological Sciences . 1994

Further evolution toward effective therapyfor acute ischemic stroke. Fisher M,Bogousslavsky J. The Journal of The American Medical Association . 1998

Neuron[P]. KUTKOVETSKYI VALENTYN YAKOVYCH;TURTI MARYNA VALENTYNIVNA.UA106289U,2016-04-25

Basic fibroblast growth factorincreases regional cerebral blood flow and reduces infarct size after experimental ischemia in a rat model. Tanaka R,Miyasaka Y,Yada K,et al. Stroke . 1995

Transient global ischemia induces dynamic changesin the expression of b F G Fand the F G Freceptor. Endoh M,Pulsinelli W A,Wagner J A. Brain Res Mol Brain Res . 1994

Endogenous neuroprotection factors and traumatic brain injury :mechanisms of action and implications for therapy. Mattson M P,Scheff S W. Journal of Neurotrauma . 1994

Basic F G Fregulates the expression of a functional 71k D N M D Areceptor protein that mediates calcium influx and neurotoxicity in hippocampal neurons. Mattson M P,Kumar K N,Wang H,et al. Journal of the Neurological Sciences . 1993

Hypotensive activity offibroblast growth factor. Cuevas P,Carceller F,Ortega S,et al. Science . 1991

Intracisternal basic fibroblast growth factor enhances functional recovery and upregulates theexpression of a molecular marker of neuronal sprouting following focalcerebral infarction. Kawamata T,Dietrich W D,Schallert T,et al. Proc Natl Acad Sci U S A . 1997

Delayed intravenous administration of basic fibroblast growth factor reduces infarct volume in a model offocal cerebral ischemia/reperfusion in the rat. Jiang N,Finklestein S P,Do T,et al. Journal of the Neurological Sciences . 1996

Usedependent growth of pyramidal neurons afterneocortical damage. Jones T A,Schallert T. Journal of the Neurological Sciences . 1994

Further evolution toward effective therapyfor acute ischemic stroke. Fisher M,Bogousslavsky J. The Journal of The American Medical Association . 1998

Neuron[P]. KUTKOVETSKYI VALENTYN YAKOVYCH;TURTI MARYNA VALENTYNIVNA.UA106289U,2016-04-25

Basic fibroblast growth factorincreases regional cerebral blood flow and reduces infarct size after experimental ischemia in a rat model. Tanaka R,Miyasaka Y,Yada K,et al. Stroke . 1995

Transient global ischemia induces dynamic changesin the expression of b F G Fand the F G Freceptor. Endoh M,Pulsinelli W A,Wagner J A. Brain Res Mol Brain Res . 1994