共 8 条
[1]
Temporal profile of neuronal damage in a modal of transient forebrain ischemia. Pulsinelli WA,Brierley JB,Plum F. Annals of Neurology . 1982
[2]
Glutamatergic control of dopamine release in the rat striatum:evidence for presynaptic N-methyl-D-Aspartate receptors on dopaminergic nerve terminals. Krebs MO,Desce JM,Kemel ML,et al. Journal of Neurochemistry . 1991
[3]
Selective neuronal vulnerability: morphologi cal and molecular characteristics. Pulsinelli WA. Progress in Brain Research . 1985
[4]
Neurology[C]. The 5th International Congress of Pathophysiology,2006
[5]
Hydrogen peroxide production by monoamine oxidase during ischemia-reperfusion in the rat brain. Simonson SG,Zhang J,Canada AT,et al. Journal of Cerebral Blood Flow and Metabolism . 1993
[6]
Effects of transientforebrain ischemia and reperfusion on function of dopaminergic neurons and dopamine reuptake in vivo in rat striatum. Akiyama Y,Ito A,Koshimura K,et al. Brain Research . 1991
[7]
Changes in extracellular concentrations of glutamate, aspartate, glycine,dopamine,serotonin and dopamine metabolites after transient global ischemia in rabbit brain. Baker AJ,Zornow MH,Scheller MS,et al. Journal of Neurochemistry . 1991
[8]
A study of the factors affecting the aluminnmoxide trihydroxyindole precedure for the analysis ofcate cholamines. Anton AH. Journal of Pharmacology and Experimental Therapeutics . 1962