瘦素及其诱导的信号传导途径

被引:9
作者
李文娟 [1 ]
陈伟 [2 ]
付小兵 [2 ]
朱任之 [1 ]
机构
[1] 兰州大学基础医学院病理研究室
[2] 解放军总医院临床部全军创伤修复重点实验室
关键词
瘦素; 受体; 信号传导;
D O I
暂无
中图分类号
R363 [病理生理学];
学科分类号
100104 ;
摘要
引用
收藏
页码:172 / 174
页数:3
相关论文
共 25 条
[1]  
Leptinenhances woundre-epitheli-alization and constitutes direct function of leptin in skin repair. Stenfan F,Birgit S,Heiko Ket al. The Journal of Clinical Investigation . 2000
[2]  
Arolefor dietaryfatinleptinreceptor,OB-Rb,function. Heshka JT,Jones PJ. Life Sciences . 2001
[3]  
Leptin receptor action in hepatic cells. Wang Y,Kuropatwinski KK,White DWet al. Journal of Biological Chemistry . 1997
[4]  
Ca2+mobilization,tyrosine hydroxylase activity,and signaling mechanisms in cultured porcine ad-renal medullary chromaffin cells:effects of leptin. Takekoshi K,Ishii K,Kawakami Yet al. The Journal of Endocrinology . 2001
[5]  
Transforming growthfactor beta1release by human adipose tissue is enhancedin obesity. Fain JN,Tichansky DS,Madan AK. Metabolism . 2005
[6]  
Proteintyrosine phospha-tase1B negatively regulatesleptin signalingin a hypothalamic cell line. Kaszubska W,Falls HD,Schaefer VGet al. Molecular and Cellular Endocrinology . 2002
[7]  
Regulation of fatty acid homeostasisin cells:novel role of leptin. Unger RH,Yhou YT,Orci L. Proceedings of the National Academy of Sciences of the United States of America . 1999
[8]  
Leptin activates the pro-moter of theinterleukin-1receptor antagonist through p42/44mitogen activated protein kinase and a composite nuclear factor kappa B/PU.1binding site. Dreyer MG,Juge-Aubry CE,Gabay Cet al. Biochemistry . 2003
[9]  
Leptininduces mitochondrial superoxide production and monocyte chemoattractant protein-1expres-sionin aortic endothelial cells byincreasingfatty acid oxidationviapro-tein kinase A. Yamagishi SI,Edelstein D,Du XLet al. Journal of Biological Chemistry . 2001
[10]  
Leptin activates Stat3,Stat1and AP-1in mouse adiposetissue. Bendinelli P,Maroni P,Pecori GFet al. Molecular and Cellular Endocrinology . 2000