类风湿关节炎骨破坏发病机制的研究进展

被引：27

作者：

韩健

张莉芸

高晋芳

马丹

机构：

[1] 山西医科大学附属大医院风湿免疫科

来源：

中国药物与临床 | 2017年 / 17卷 / 05期

关键词：

D O I：

暂无

中图分类号：

R593.22 [类风湿性关节炎];

学科分类号：

100201 [内科学];

摘要：

<正>类风湿关节炎(rheumatoid arthritis,RA)是临床常见的慢性全身性自身免疫性疾病。发病机制为免疫紊乱导致滑膜炎及侵袭性新生血管翳形成,晚期往往会引发软骨及骨的破坏,最终导致严重的关节畸形甚至残疾。但至今RA仍得不到完全根治,原因在于其确切病因尤其是骨破坏的发病机制至今尚未完全阐明。

引用

页码：674 / 677

页数：4

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