重视对幽门螺杆菌感染临床结局多样性的研究

被引：36

作者：

萧树东

刘文忠

机构：

[1] 上海第二医科大学附属仁济医院、上海市消化疾病研究所,上海第二医科大学附属仁济医院、上海市消化疾病研究所

来源：

中华内科杂志 | 1999年 / 07期

关键词：

临床结局; 基因型; 遗传型; 致病岛; 毒力因子; 胃黏膜上皮细胞;

D O I：

暂无

中图分类号：

R378 [病原细菌];

学科分类号：

100103 ; 100705 ;

摘要：

引用

页码：4 / 5

页数：2

共 7 条

[1]

RegulationofgastricepithelialcellgrowthbyHelicobacterpylori:evidenceforamajorroleofapopto sis. WangerS,BeilW,WestermannJ,etal. Gastroenterology . 1997

[2]

ClinicalandpathologicalimportanceofheterogeneityinvacA ,thevacuolatingcytotoxingeneofHelicobacterpylori. AthertonJC,PeekRMJr,ThamKT,etal. Gastroenterology . 1997

[3]

AanalysesofthecagpathogenicityislandofHelicobacterpylori. CliftonSW,KersulyteD,etal. Molecular Microbiology . 1998

[4]

EvidenceofnovelpathogenicpathwaysfortheformationofantigastricautoantibodiesinHelicobacterpylorigastritis. FallerG,SteiningerH,AppelmelkB,etal. Journal of Clinical Pathology . 1998

[5]

TheroleoftheHLA DQA1geneinresis tancetoatrophicgastritisandgastricadenocarcinomainducedbyHeli cobacterpyloriinfection. AzumaT,ItoS,SatoF,etal. Cancer . 1998

[6]

Cag :apathogenicityislandofHelicobacterpylori,encodestypeIspecificanddiseaseassociatedviru lencefactors. CensiniS,LangeC,XiangZY,etal. Proceedings of the National Academy of Sciences of the United States of America . 1996

[7]

ThegastricH + K + ATPaseisamajorautoantigeninchronicHelicobacterpylorigastritiswithbodymucosaatrophy. ClaeysD,FallerG,AppelmelkBJ,etal. Gastroenterology . 1998

← 1 →