共 19 条
[1]
Asociation cortex,cerebellum and serum concentrations C1q and factor B in Alzheimer’s disease. Brachova L,Lue LF,Schultz J,et al. Molecular Brain Research . 1993
[2]
Immunohistochemical location of vitronectin, its receptor and beta-3 integrin in Alzheimer brain tissue. Akiyama H,Kwamata T,Dedhar S,et al. J Neuroimmunol . 1991
[3]
Serum amyloid P component binds to histones and activates the classical component pathway. Hicks PS,Sunero-Nazia L,Duclos TW,et al. J Immunol . 1992
[4]
Molecular aspects of inflammatory and immune responses in Alzheimer’s disease. Kalaria RN,Harshbarger-Kelly DL,Cohen DL,et al. Neurobiology of Aging . 1996
[5]
Complement gene expressionin human brain : comparison between normal and Alzheimer’s disease cases. Walker DG,McGeer PL. Molecular Brain Research . 1992
[6]
Complement C1 inhibitor is produced by brain tissue and is elevated in Alzheimer’s disease. Walker DG,Yasuhara O,Patston PA,et al. Brain Research . 1995
[7]
Scavanger recepter mediated adhension of microglia toβ-amyloid fibrils. Khoury EJ,Hickman SE,Thomas CA,et al. Nature . 1996
[8]
The role of complement and activated microglia in the pathogenesis of Alzheimer’s disease. Eikelenboom P,Veerhuis R. Neurobiology of Aging . 1996
[9]
Charge-based binding of complement component C1q to the Alzheimer ’ s disease. Webster S,Bonne LL,Rogers J. American Journal of Pathology . 1997
[10]
Autoantibody reactivity in serum of patients with Alzheimer ’ s disease and other age-related dementias. Scott K,McGeer ER. Psychiatry Research . 1996