急性脑创伤后继发性神经细胞损伤的研究进展

[1] Pathophysiology of neural injury: therapeutic opportunities and challenges. Povlishock JT. Clinical Neurosurgery . 2000

[2] Pharmacologicaltreatmentoftraumaticbraininjuryareviewofagentsindevelopment. HattonJ. CNS Drugs . 2001

[3] Ca2+-Mediated Mitochondrial Dysfunction and the Protective Effects of Bcl-2. Murphy AN. Annals of the New York Academy of Sciences . 1999

[4] Neuronal cell death in nervous system development, disease, and injury (Review). Martin LJ. International Journal of Molecular Medicine . 2001

[5] Traumatic injury induces differential expression of cell death genes in organotypic brain slice cultures determined by complementary DNA array hybridization. Morrison B,Eberwine JH,Meaney DF, et al. Neuroscience . 2000

[6] An overview of new and novel pharmacotherapies for use in traumatic brain injury. Vink,R,Nimmo,AJ,Cernak,I. Clinical and Experimental Pharmacology and Physiology . 2001

[7] Cerebral cortical neuron apoptosis after mild excitotoxic injury in vitro: different roles of mesencephalic and cortical astrocytes. Huang PP, Esquenazi S, Le RPD. Neurosurgery . 1999

[8] Neurology[C]. The 5th International Congress of Pathophysiology,2006

[9] Apoptoticandantiapoptoticmechanismsinstroke. MattsonMP. Cell and Tissue Research . 2000

[10] Low -affinity kainate receptor agonists induce insult - dependent apoptosis and necrosis in cultured murine cortical neurons. Moldrich RX, Beart PM, Pascoe CJ, et al. Journal of Neuroscience Research . 2000

[1] Pathophysiology of neural injury: therapeutic opportunities and challenges. Povlishock JT. Clinical Neurosurgery . 2000

[2] Pharmacologicaltreatmentoftraumaticbraininjuryareviewofagentsindevelopment. HattonJ. CNS Drugs . 2001

[3] Ca2+-Mediated Mitochondrial Dysfunction and the Protective Effects of Bcl-2. Murphy AN. Annals of the New York Academy of Sciences . 1999

[4] Neuronal cell death in nervous system development, disease, and injury (Review). Martin LJ. International Journal of Molecular Medicine . 2001

[5] Traumatic injury induces differential expression of cell death genes in organotypic brain slice cultures determined by complementary DNA array hybridization. Morrison B,Eberwine JH,Meaney DF, et al. Neuroscience . 2000

[6] An overview of new and novel pharmacotherapies for use in traumatic brain injury. Vink,R,Nimmo,AJ,Cernak,I. Clinical and Experimental Pharmacology and Physiology . 2001

[7] Cerebral cortical neuron apoptosis after mild excitotoxic injury in vitro: different roles of mesencephalic and cortical astrocytes. Huang PP, Esquenazi S, Le RPD. Neurosurgery . 1999

[8] Neurology[C]. The 5th International Congress of Pathophysiology,2006

[9] Apoptoticandantiapoptoticmechanismsinstroke. MattsonMP. Cell and Tissue Research . 2000

[10] Low -affinity kainate receptor agonists induce insult - dependent apoptosis and necrosis in cultured murine cortical neurons. Moldrich RX, Beart PM, Pascoe CJ, et al. Journal of Neuroscience Research . 2000