<正> To explore the influence and significance of the ischemia reperfusion on the hepatic neoplasm, the hepaticVX2 neoplasm model of rabbits was established under the guide of ultrasonography; and ischemia was caused by usinga non-traumatic vascular clamp to block the branches distributing in the left-middle lobe of the hepatic artery for 60min, and subsequently the clamp was removed and the reperfusion injury of hepatic neoplasm occurred. At differenttime-points, the normal and hepatic neoplasm tissues of the animal models were taken out to detect the superoxide dis-mutase (SOD) and malondialdehyde (MDA) respectively. The results show that the products and injurious effects ofoxygen free radical (OFR) of the neoplasm tissues are more serious than those of the normal hepatic tissues.
