共 18 条
[1]
Neuroimmune mechanisms in Alzheimer, s disease pathogenesis. McGeer PL,Rogers J,McGeer EG. Alzheimer’s Dis Assoc Dis . 1994
[2]
Immunohistochemical study of theβchemokine receptors CCR3 and CCR5 and their ligands in normal and Alzheimer’s disease brains. Xia MQ,Qin SX,Wu LJ,et al. American Journal of Pathology . 1998
[3]
Chronic overproduction of transforming growth factor-β1 by astrocytes promotes Alzheimer’s disease-like microvascular degeneration in transgenic mice. Wyss-Coray T,Lin C,Sanan DA,et al. American Journal of Pathology . 2000
[4]
Microglial activation in Alzheimer disease: Association with APOE genotype. Egensperger R,Kosel S,von Eitzen U,et al. Brain Pathology . 1998
[5]
Cytokines associated with amyloid plaques in Alzheimer’s disease brain stimulate human glial and neuronal cell cultures to secrete early complement proteins, but not C1-inhibitor. Veerhuis R,Janssen I,De Groot CJ,et al. Experimental Neurology . 1999
[6]
Temporal accrual of complement proteins in amyloid plaques in Down’s syndrome with Alzheimer’s disease. Stoltzner SE,Grenfell TJ,Mori C,et al. American Journal of Pathology . 2000
[7]
Oncostatin M and the interleukin-6 and soluble interleukin-6 receptor complex regulateα1-antichymotrypsin expression in human cortical astrocytes. Kordula T,Rydel RE,Brigham EF,et al. Journal of Biological Chemistry . 1998
[8]
Glial-derived proteins activate cultured astrocytes and enhance βamyloid-induced glial activation. Hu J,Van Eldik LJ. Brain Research . 1999
[9]
β amyloidactivatestheO- 2formingNADPHoxidaseinmicroglia,monocytes,andneutrophils.ApossibleinflammatorymechanismofneuronaldamageinAlzheimer’’sdisease. BiancaVD,DusiS,BianchiniE ,etal. Biochemical Journal . 1999
[10]
Themicroglialcell:Ahistoricalreview. BarronKD. JNeurolSci . 1995