银屑病发病机制研究进展

被引:54
作者
马天
姜文成
陈洁
李斌
机构
[1] 上海中医药大学附属岳阳中西医结合医院皮肤科
基金
上海市自然科学基金;
关键词
银屑病; 白细胞介素23; CD4阳性T淋巴细胞;
D O I
暂无
中图分类号
R758.63 [牛皮癣(银屑病)];
学科分类号
100227 [皮肤病学];
摘要
银屑病(psoriasis)是多种免疫细胞共同参与的慢性复发性炎症性皮肤病,它具有明显的鳞屑性红斑和不同程度的自觉症状,严重影响患者的生活质量。该病的发病机制探讨多集中在遗传、感染、免疫等方面。目前,越来越多的研究证明了IL-23和CD4阳性辅助性T淋巴细胞17(CD4+T helper 17,Th17)在银屑病病理机制中起到了关键作用。本文就银屑病发病机制的进展作一综述。
引用
收藏
页码:9656 / 9658
页数:3
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