桂枝挥发油的药理作用研究进展

被引：351

作者：

徐锋

王德健

王凤

温桃群

桑文涛

曾南

机构：

[1] 成都中医药大学药学院中药材标准化教育部重点实验室四川省中药资源系统研究与开发利用重点实验室——省部共建国家重点实验室培育基地

来源：

中华中医药杂志 | 2016年 / 31卷 / 11期

关键词：

桂枝; 挥发油; 桂皮醛; 抗炎; 抗病毒; 抗肿瘤; 抗菌;

D O I：

暂无

中图分类号：

R285 [中药药理学];

学科分类号：

100806 [中药药理学];

摘要：

近年来对临床常用中药桂枝的研究发现,挥发油为桂枝的主要活性部位群,故桂枝挥发油的药理研究获得广泛关注。文章就桂枝挥发油药理作用的研究现状进行归纳、分析,主要包括抗炎、抗病毒、抗肿瘤和抗菌等方面,研究结果为桂枝的功效与临床应用提供了药理学依据,亦为桂枝挥发油的进一步研究、开发及应用提供参考。

引用

页码：4653 / 4657

页数：5

共 55 条

[1]

Protective effects of cinnamic acid and cinnamic aldehyde on isoproterenol-induced acute myocardial ischemia in rats [J].

Song, Fan ;

Li, Hua ;

Sun, Jiyuan ;

Wang, Siwang .

JOURNAL OF ETHNOPHARMACOLOGY, 2013, 150 (01) :125-130

[2]

Cinnamaldehyde protects from the hypertension associated with diabetes [J].

El-Bassossy, Hany M. ;

Fahmy, Ahmed ;

Badawy, Dina .

FOOD AND CHEMICAL TOXICOLOGY, 2011, 49 (11) :3007-3012

[3]

The cinnamon-derived Michael acceptor cinnamic aldehyde impairs melanoma cell proliferation; invasiveness; and tumor growth.[J].Christopher M. Cabello;Warner B. Bair;Sarah D. Lamore;Stephanie Ley;Alexandra S. Bause;Sara Azimian;Georg T. Wondrak.Free Radical Biology and Medicine.2008, 2

[4]

Cinnamaldehyde suppresses toll-like receptor 4 activation mediated through the inhibition of receptor oligomerization.[J].Hyung S. Youn;Jun K. Lee;Yong J. Choi;Shin I. Saitoh;Kensuke Miyake;Daniel H. Hwang;Joo Y. Lee.Biochemical Pharmacology.2007, 2

[5]

Cinnamaldehyde inhibits pro-inflammatory cytokines secretion from monocytes/macrophages through suppression of intracellular signaling.[J].Louis Kuoping Chao;Kuo-Feng Hua;Hsien-Yeh Hsu;Sen-Sung Cheng;I-Fan Lin;Chia-Jung Chen;Shui-Tein Chen;Shang-Tzen Chang.Food and Chemical Toxicology.2007, 1

[6]

Inhibitory effect of cinnamaldehyde, derived from Cinnamomi cortex, on the growth of influenza A/PR/8 virus in vitro and in vivo [J].

Hayashi, K. ;

Imanishi, N. ;

Kashiwayama, Y. ;

Kawano, A. ;

Terasawa, K. ;

Shimada, Y. ;

Ochiai, H. .

ANTIVIRAL RESEARCH, 2007, 74 (01) :1-8

[7]

Suppression of age-related inflammatory NF-κB activation by cinnamaldehyde [J].

Kim, Dae Hyun ;

Kim, Chul Hong ;

Kim, Min-Sun ;

Kim, Ji Young ;

Jung, Kyung Jin ;

Chung, Jae Heun ;

An, Won Gun ;

Lee, Jae Won ;

Yu, Byung Pal ;

Chung, Hae Young .

BIOGERONTOLOGY, 2007, 8 (05) :545-554

[8]

Antimutagenicity of cinnamaldehyde and vanillin in human cells: Global gene expression and possible role of DNA damage and repair.[J].Audrey A. King;Daniel T. Shaughnessy;Kanae Mure;Joanna Leszczynska;William O. Ward;David M. Umbach;Zongli Xu;Danica Ducharme;Jack A. Taylor;David M. DeMarini;Catherine B. Klein.Mutation Research - Fundamental and Molecular Mechanisms of Mutagenesis.2006, 1

[9]

Cinnamaldehyde-induced apoptosis in human PLC/PRF/5 cells through activation of the proapoptotic Bcl-2 family proteins and MAPK pathway [J].