砷化物所致细胞恶性转化的信号通路研究进展

[1] 砷诱导NIH3T3细胞转化模型的建立 [J]. 新疆医科大学学报, 2004, (01) : 1 - 5

[2] NIH 3T3 cells malignantly transformed by mot-2 showinactivation and cytoplasmic sequestration of the p53protein[J]. WADHIVA RENU;SYIJICHI TAKANO;YOUJIMITSUI;SUNIL C KAUL( Chugai Research Institute for Molecular Medicine,153-2 Nagai, Niihari-Mura, Ibaraki 300-41, Japan)( National Institute of Bioscience and Hunan-Technology, AIST, 1-1 Higashi, Tsukusa, Ibaraki 305-85.Cell Research. 1999(04)

[3] mot-2-Mediated Cross Talk between Nuclear Factor-[kappa]B and p53 Is Involved in Arsenite-Induced Tumorigenesis of Human Embryo Lung Fibroblast Cells[J] . Li, Yuan,Xu, Yuan,Ling, Min,Yang, Ye,Wang, Shoulin,Li, Zhong,Zhou, Jianwei,Wang, Xinru,Liu, Qizhan.Environmental Health Perspectives . 2010 (7)

[4] Arsenic-induced malignant transformation of human keratinocytes: Involvement of Nrf2[J] . Jingbo Pi,Bhalchandra A. Diwan,Yang Sun,Jie Liu,Wei Qu,Yuying He,Miroslav Styblo,Michael P. Waalkes.Free Radical Biology and Medicine . 2008 (5)

[5] PI-3K/Akt Pathway-Dependent Cyclin D1 Expression Is Responsible for Arsenite-Induced Human Keratinocyte Transformation[J] . Ouyang, Weiming,Luo, Wenjing,Zhang, Dongyun,Jian, Jinlong,Ma, Qian,Li, Jingxia,Shi, Xianglin,Chen, Jingyuan,Gao, Jimin,Huang, Chuanshu.Environmental Health Perspectives . 2008 (1)

[6] Phosphorylation of CBP by IKKα promotes cell growth by switching the binding preference of CBP from p53 to NF-κB [J]. MOLECULAR CELL, 2007, 26 (01) : 75 - 87

[7] Biphasic effect of arsenite on cell proliferation and apoptosis is associated with the activation of JNK and ERK1/2 in human embryo lung fibroblast cells [J]. TOXICOLOGY AND APPLIED PHARMACOLOGY, 2007, 220 (01) : 18 - 24

[8] PI-3K/Akt signal pathway plays a crucial role in arsenite-induced cell proliferation of human keratinocytes through induction of cyclin D1 [J]. JOURNAL OF CELLULAR BIOCHEMISTRY, 2007, 101 (04) : 969 - 978

[9] Molecular mechanisms of arsenic carcinogenesis [J]. Molecular and Cellular Biochemistry, 2004, 255 : 57 - 66

[10] Arsenic Disrupts Cellular Levels of p53 and mdm2: A Potential Mechanism of Carcinogenesis[J] . Hisham K. Hamadeh,Maricelly Vargas,Edward Lee,Daniel B. Menzel.Biochemical and Biophysical Research Communications . 1999 (2)

[1] 砷诱导NIH3T3细胞转化模型的建立 [J]. 新疆医科大学学报, 2004, (01) : 1 - 5

[2] NIH 3T3 cells malignantly transformed by mot-2 showinactivation and cytoplasmic sequestration of the p53protein[J]. WADHIVA RENU;SYIJICHI TAKANO;YOUJIMITSUI;SUNIL C KAUL( Chugai Research Institute for Molecular Medicine,153-2 Nagai, Niihari-Mura, Ibaraki 300-41, Japan)( National Institute of Bioscience and Hunan-Technology, AIST, 1-1 Higashi, Tsukusa, Ibaraki 305-85.Cell Research. 1999(04)

[3] mot-2-Mediated Cross Talk between Nuclear Factor-[kappa]B and p53 Is Involved in Arsenite-Induced Tumorigenesis of Human Embryo Lung Fibroblast Cells[J] . Li, Yuan,Xu, Yuan,Ling, Min,Yang, Ye,Wang, Shoulin,Li, Zhong,Zhou, Jianwei,Wang, Xinru,Liu, Qizhan.Environmental Health Perspectives . 2010 (7)

[4] Arsenic-induced malignant transformation of human keratinocytes: Involvement of Nrf2[J] . Jingbo Pi,Bhalchandra A. Diwan,Yang Sun,Jie Liu,Wei Qu,Yuying He,Miroslav Styblo,Michael P. Waalkes.Free Radical Biology and Medicine . 2008 (5)

[5] PI-3K/Akt Pathway-Dependent Cyclin D1 Expression Is Responsible for Arsenite-Induced Human Keratinocyte Transformation[J] . Ouyang, Weiming,Luo, Wenjing,Zhang, Dongyun,Jian, Jinlong,Ma, Qian,Li, Jingxia,Shi, Xianglin,Chen, Jingyuan,Gao, Jimin,Huang, Chuanshu.Environmental Health Perspectives . 2008 (1)

[6] Phosphorylation of CBP by IKKα promotes cell growth by switching the binding preference of CBP from p53 to NF-κB [J]. MOLECULAR CELL, 2007, 26 (01) : 75 - 87

[7] Biphasic effect of arsenite on cell proliferation and apoptosis is associated with the activation of JNK and ERK1/2 in human embryo lung fibroblast cells [J]. TOXICOLOGY AND APPLIED PHARMACOLOGY, 2007, 220 (01) : 18 - 24

[8] PI-3K/Akt signal pathway plays a crucial role in arsenite-induced cell proliferation of human keratinocytes through induction of cyclin D1 [J]. JOURNAL OF CELLULAR BIOCHEMISTRY, 2007, 101 (04) : 969 - 978

[9] Molecular mechanisms of arsenic carcinogenesis [J]. Molecular and Cellular Biochemistry, 2004, 255 : 57 - 66

[10] Arsenic Disrupts Cellular Levels of p53 and mdm2: A Potential Mechanism of Carcinogenesis[J] . Hisham K. Hamadeh,Maricelly Vargas,Edward Lee,Daniel B. Menzel.Biochemical and Biophysical Research Communications . 1999 (2)