心境障碍的神经生物学研究进展

[1] Increased hippocampal BDNF immunoreactivity in subjects treated with antidepressant medication. Chen B,Dowlatshahi D,MacQueen GM. Biological Psychiatry . 2001

[2] Glial reduction in the subgenual prefrontal cortex in mood disorders. Ongur D,Drevets WC,Price JL. Proceedings of the National Academy of Sciences of the United States of America . 1998

[3] Signaling: cellular insights into the pathophysiology of bipolar disorder. Manji,H,Lenox,RH. Biological Psychiatry . 1999

[4] Histone deacetylase is a direct target of valproic acid, a potent anticonvulsant, mood stabilizer,and teratogen. Phiel C,Zhang F,Huang E,et al. Biochemical Journal . 2001

[5] Neuroprotective effects of lithium in cultured cells and animal models of diseases. Chuang DM,Chen RW,Chalecka-Franaszek E,et al. Bipolar Disorders . 2002

[6] Neuronal plasticity and survival in mood disorders. Duman R,Malberg J,Nakagawa S,et al. Biol Psychiaty . 2000

[7] BDNF-mediated signal transduction is modulated by GSK3beta and mood stabilizing agents. Mai L,Jope RS,Li X. Journal of Neurochemistry . 2002

[8] Changes in the pattern of brain-derived neurotrophic factor immunoreactivity in the rat brain after acute and subchronic haloperidol treatment. Dawson N,Hamid EH,Egan MF,et al. Synapse . 2001

[9] Neurobiologyofmentalill ness. DrevetsWC ,,GaddeK,KrishnanR. . 1999

[10] The mood stabilizer valproic acid activates mitogen-activated protein kinases and promotes neurite growth. Yuan P,Juong LD,Jiang YM,et al. Biological Chemistry . 2001

[1] Increased hippocampal BDNF immunoreactivity in subjects treated with antidepressant medication. Chen B,Dowlatshahi D,MacQueen GM. Biological Psychiatry . 2001

[2] Glial reduction in the subgenual prefrontal cortex in mood disorders. Ongur D,Drevets WC,Price JL. Proceedings of the National Academy of Sciences of the United States of America . 1998

[3] Signaling: cellular insights into the pathophysiology of bipolar disorder. Manji,H,Lenox,RH. Biological Psychiatry . 1999

[4] Histone deacetylase is a direct target of valproic acid, a potent anticonvulsant, mood stabilizer,and teratogen. Phiel C,Zhang F,Huang E,et al. Biochemical Journal . 2001

[5] Neuroprotective effects of lithium in cultured cells and animal models of diseases. Chuang DM,Chen RW,Chalecka-Franaszek E,et al. Bipolar Disorders . 2002

[6] Neuronal plasticity and survival in mood disorders. Duman R,Malberg J,Nakagawa S,et al. Biol Psychiaty . 2000

[7] BDNF-mediated signal transduction is modulated by GSK3beta and mood stabilizing agents. Mai L,Jope RS,Li X. Journal of Neurochemistry . 2002

[8] Changes in the pattern of brain-derived neurotrophic factor immunoreactivity in the rat brain after acute and subchronic haloperidol treatment. Dawson N,Hamid EH,Egan MF,et al. Synapse . 2001

[9] Neurobiologyofmentalill ness. DrevetsWC ,,GaddeK,KrishnanR. . 1999

[10] The mood stabilizer valproic acid activates mitogen-activated protein kinases and promotes neurite growth. Yuan P,Juong LD,Jiang YM,et al. Biological Chemistry . 2001