共 12 条
[1]
Resistance of alpha-synuclein null mice to the parkinsonian neurotoxin MPTP. Dauer W,Kholodilov N,Vila M,et al. Proceedings of the National Academy of Sciences of the United States of America . 2002
[2]
Parkin localizes to the Lewy bodies of Parkinson disease and dementia with Lewy bodies. Schlossmacher MG,Frosch MP,Gai WP,et al. American Journal of Pathology . 2002
[3]
Subcutaneous rotenone exposure causes highly selective dopaminergic degeneration and alpha-synuclein aggregation. Sherer TB,Kim JH,Betarbet R,et al. Experimental Neurology . 2003
[4]
The N-terminal repeat domain of alpha-synuclein inhibits beta-sheet and amyloid fibril formation. Kessler JC,Rochet JC,Lansbury PT Jr. Biochemistry . 2003
[5]
Inducible expression of mutant alpha-synuclein decreases proteasome activity and increases sensitivity to mitochondria-dependent apoptosis. Tanaka Y,Engelender S,Igarashi S,et al. Human Molecular Genetics . 2001
[6]
Human alpha-synuclein over-expression increases intracellular reactive oxygen species levels and susceptibility to dopamine. Junn E,Mouradian MM. Neuroscience Letters . 2002
[7]
Conformational properties of alpha-synuclein in its free and lipid-associated states. Eliezer D,Kutluay E,Bussell R Jr,et al. Journal of Molecular Biology . 2001
[8]
Proteasomal inhibition leads to formation of ubiquitin/alpha-synuclein-immunoreactive inclusions in PC12 cells. Rideout HJ,Larsen KE,Sulzer D,et al. Journal of Neurochemistry . 2001
[9]
Apoptotic signaling in dopamine-induced cell death: the role of oxidative stress, p38 mitogen-activated protein kinase, cytochrome c and caspases. Junn E,Mouradian MM. Journal of Neurochemistry . 2001
[10]
Proteasome inhibition causes nigral degeneration with inclusion bodies in rats. McNaught KS,Bjorklund LM,Belizaire R,et al. NeuroReport . 2002