共 18 条
[1]
Intravenousimmunoglobu-lintherapy induces neutrophil apoptosis in kawasaki disease. Tsujimoto H,Takeshita S,Nakatani K,et al. Clinical Immunology . 2002
[2]
intravenousimmunoglobulin pre-vents experimental autoimmune myositis in SJL mice by reducing anti-myosin antibody and by blocking complement deposition. Wada J,Shintani N,Kikutani K,et al. Clinical and Experimental Immunology . 2001
[3]
Anti-cytokine nature of natural humanimmunoglobulin:one possible mechanismof the clinical effect of intravenousimmunoglobulintherapy. Abe Y,horiuchi A,Miyake M,et al. Immunological Reviews . 1994
[4]
The B cell su-perantigen-like interaction of intravenous immunoglobin(IVIg)with Fabfragments of V(H)3-23and3-30/3-30.5germline gene origin cloned from a patient with Kawasaki disease is enhanced after IVIg therapy. Leucht S,Uttenreuther-Fischer MM,Gaedicke G,et al. Clinical Immunology . 2001
[5]
The B cell su-perantigen-like interaction of intravenous immunoglobin(IVIg)with Fabfragments of V(H)3-23and3-30/3-30.5germline gene origin cloned from a patient with Kawasaki disease is enhanced after IVIg therapy. Leucht S,Uttenreuther-Fischer MM,Gaedicke G,et al. Clinical Immunology . 2001
[6]
Human polyspecific immunoglobulin for therapeutic use induces p21/WAF-1and Bcl-2,which may byresponsible for G1arrest andlong-termsurvival. Ekberg C,Nordstrom E,Skansen-Saphir U,et al. Human Immunology . 2001
[7]
Pooled human IgG modulates cytokine production in lymphocytes and monocytes. Andersson U,Bjork L,Skansen-Saphir U,et al. Immunological Reviews . 1994
[8]
Anti-inflammatory activity of IVIg mediated through the inhibitory Fc receptor. Samuelssion A,Towers TL,Ravetch JV. Science . 2001
[9]
Polyspecific immunoglobulins(IVIg)suppress proliferation of human(auto)antigen-specific Tcells withoutinducing apoptosis. Aktas O,Waiczies S,Grieger U,et al. J Neuroimmunol . 2001
[10]
Inhibition of IgE production in vitro by intact and fragmented intravenous immunoglubulin. Zhuang Q,Mazer B. The Journal of Allergy and Clinical Immunology . 2001