MCP-1 induces a novel transcription factor with proapoptotic activity

被引：27

作者：

Bidzhekov, Kiril ^{[1
]}

Zernecke, Alma ^{[1
]}

Weber, Christian ^{[1
]}

机构：

[1] RWTH Aachen Univ Hosp, Inst Mol Cardiovasc Res, Aachen, Germany

来源：

CIRCULATION RESEARCH | 2006年 / 98卷 / 09期

关键词：

chemokines; apoptosis; transcription factors; ischemic heart disease;

D O I：

10.1161/01.RES.0000223483.12225.80

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：1107 / 1109

页数：3

共 25 条

[1] Elevated circulating levels of C-C chemokines in patients with congestive heart failure [J].

Aukrust, P ;

Ueland, T ;

Müller, F ;

Andreassen, AK ;

Nordoy, I ;

Aas, H ;

Kjekshus, J ;

Simonsen, S ;

Froland, SS ;

Gullestad, L .

CIRCULATION, 1998, 97 (12) :1136-1143

[2] Monocyte chemoattractant protein-1 is upregulated in rats with volume-overload congestive heart failure [J].

Behr, TM ;

Wang, XK ;

Aiyar, N ;

Coatney, RW ;

Li, X ;

Koster, P ;

Angermann, CE ;

Ohlstein, E ;

Feuerstein, GZ ;

Winaver, J .

CIRCULATION, 2000, 102 (11) :1315-1322

[3] Decreased lesion formation in CCR2-/- mice reveals a role for chemokines in the initiation of atherosclerosis [J].

Boring, L ;

Gosling, J ;

Cleary, M ;

Charo, IF .

NATURE, 1998, 394 (6696) :894-897

[4] Chemokines in the pathogenesis of vascular disease [J].

Charo, IF ;

Taubman, MB .

CIRCULATION RESEARCH, 2004, 95 (09) :858-866

[5] Association between plasma levels of monocyte chemoattractant protein-1 and long-term clinical outcomes in patients with acute coronary syndromes [J].

de Lemos, JA ;

Morrow, DA ;

Sabatine, MS ;

Murphy, SA ;

Gibson, CM ;

Antman, EM ;

McCabe, CH ;

Cannon, CP ;

Braunwald, E .

CIRCULATION, 2003, 107 (05) :690-695

[6] CCL2/monocyte chemoattractant protein-1 regulates inflammatory responses critical to healing myocardial infarcts [J].

Dewald, O ;

Zymek, P ;

Winkelmann, K ;

Koerting, A ;

Ren, GF ;

Abou-Khamis, T ;

Michael, LH ;

Rollins, BJ ;

Entman, ML ;

Frangogiannis, NG .

CIRCULATION RESEARCH, 2005, 96 (08) :881-889

[7] Death begets failure in the heart [J].

Foo, RSY ;

Mani, K ;

Kitsis, RN .

JOURNAL OF CLINICAL INVESTIGATION, 2005, 115 (03) :565-571

[8] Critical role for monocyte chemoattractant protein-1 and macrophage inflammatory protein-1 α in induction of experimental autoimmune myocarditis and effective anti-monocyte chemoattractant protein-1 gene therapy [J].

Göser, S ;

Öttl, R ;

Brodner, A ;

Dengler, TJ ;

Torzewski, J ;

Egashira, K ;

Rose, NR ;

Katus, HA ;

Kaya, Z .

CIRCULATION, 2005, 112 (22) :3400-3407

[9] Absence of monocyte chemoattractant protein-1 reduces atherosclerosis in low density lipoprotein receptor-deficient mice [J].

Gu, L ;

Okada, Y ;

Clinton, SK ;

Gerard, C ;

Sukhova, GK ;

Libby, P ;

Rollins, BJ .

MOLECULAR CELL, 1998, 2 (02) :275-281

[10] Anti-monocyte chemoattractant protein-1 gene therapy attenuates left ventricular remodeling and failure after experimental myocardial infarction [J].

Hayashidani, S ;

Tsutsui, H ;

Shiomi, T ;

Ikeuchi, M ;

Matsusaka, H ;

Suematsu, N ;

Wen, J ;

Egashira, K ;

Takeshita, A .

CIRCULATION, 2003, 108 (17) :2134-2140

← 1 2 3 →