Presenilins, processing of β-amyloid precursor protein, and Notch signaling

被引：57

作者：

Chan, YM ^{[1
]}

Jan, YN ^{[1
]}

机构：

[1] Univ Calif San Francisco, Howard Hughes Med Inst, Dept Physiol, Dept Biochem, San Francisco, CA 94143 USA

来源：

NEURON | 1999年 / 23卷 / 02期

关键词：

D O I：

10.1016/S0896-6273(00)80771-0

中图分类号：

Q189 [神经科学];

学科分类号：

071006 ;

摘要：

[No abstract available]

引用

页码：201 / 204

页数：4

共 19 条

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[7] HOP-1, a Caenorhabditis elegans presenilin, appears to be functionally redundant with SEL-12 presenilin and to facilitate LIN-12 and GLP-1 signaling
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[8] ANTINEUROGENIC PHENOTYPES INDUCED BY TRUNCATED NOTCH PROTEINS INDICATE A ROLE IN SIGNAL-TRANSDUCTION AND MAY POINT TO A NOVEL FUNCTION FOR NOTCH IN NUCLEI
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