Conditional inactivation of Sox9:: A mouse model for campomelic dysplasia

被引：94

作者：

Kist, R

Schrewe, H

Balling, R

Scherer, G

机构：

[1] Univ Freiburg, Inst Human Genet, D-79106 Freiburg, Germany

[2] Univ Freiburg, Fac Biol, D-79106 Freiburg, Germany

[3] GSF, Natl Res Ctr Environm & Hlth, Inst Mammalian Genet, Neuherberg, Germany

[4] Max Planck Inst Immunobiol, Freiburg, Germany

来源：

GENESIS | 2002年 / 32卷 / 02期

关键词：

D O I：

10.1002/gene.10050

中图分类号：

Q [生物科学];

学科分类号：

07 ; 0710 ; 09 ;

摘要：

[No abstract available]

引用

页码：121 / 123

页数：3

共 16 条

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[2] Sox9 is required for cartilage formation
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[3] CAMPOMELIC DYSPLASIA AND AUTOSOMAL SEX REVERSAL CAUSED BY MUTATIONS IN AN SRY-RELATED GENE
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[4] DNA-BINDING PROPERTIES OF THE HMG DOMAIN OF THE LYMPHOID-SPECIFIC TRANSCRIPTIONAL REGULATOR LEF-1
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[5] HOUSTON CS, 1988, AM J MED GENET, V15, P3
[6] Conditional gene knockout using Cre recombinase
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[7] A CLINICAL AND GENETIC-STUDY OF CAMPOMELIC DYSPLASIA
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[8] Nagy A, 2000, GENESIS, V26, P99, DOI 10.1002/(SICI)1526-968X(200002)26:2<99::AID-GENE1>3.0.CO
[9] 2-B
[10] SOX9 binds DNA, activates transcription, and coexpresses with type II collagen during chondrogenesis in the mouse
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[J]. DEVELOPMENTAL BIOLOGY, 1997, 183 (01) : 108 - 121

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